PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15935814-7	2005	We have found that both wild type and AhR deficient cells are sensitive to catechol and hydroquinone-initiated increases in c-Myb activity while both cell types are resistant to benzene-initiated alterations leaving the role of the AhR still undetermined.	catechol	75-83	aryl-hydrocarbon receptor	Mus musculus	38-41	
15935814-8	2005	Interestingly, protein expression of c-Myb is increased after catechol exposure in AhR deficient cells while decreased in wild-type cells.	catechol	62-70	aryl-hydrocarbon receptor	Mus musculus	83-86