PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34911733-7	2022	Finally, biochemical experiments indicate that luminal-like DTPs survive via estrogen receptor-dependent induction of SGK3, leading to rewiring of the PI3K/AKT/mTORC1 pathway to enable AKT-independent mTORC1 activation.	Phenobarbital	47-54	AKT serine/threonine kinase 1	Homo sapiens	156-159	
34911733-7	2022	Finally, biochemical experiments indicate that luminal-like DTPs survive via estrogen receptor-dependent induction of SGK3, leading to rewiring of the PI3K/AKT/mTORC1 pathway to enable AKT-independent mTORC1 activation.	Phenobarbital	47-54	AKT serine/threonine kinase 1	Homo sapiens	185-188	
34314419-8	2021	In contrast, AKT activation altered the ERG cistrome and promoted expression of luminal cell fate genes.	Phenobarbital	80-87	AKT serine/threonine kinase 1	Homo sapiens	13-16	
33009488-0	2020	De novo induction of lineage plasticity from human prostate luminal epithelial cells by activated AKT1 and c-Myc.	Phenobarbital	60-67	AKT serine/threonine kinase 1	Homo sapiens	98-102	
31991499-11	2020	c-Met downstream pathway genes (RAS-MAPK, PI3K-AKT and angiogenesis pathway) showed significant upregulation in Luminal B molecular subtype, lymph node-positive cases and cases with high MVD.	Phenobarbital	112-119	AKT serine/threonine kinase 1	Homo sapiens	47-50	
33024416-16	2020	The final results indicated that LINC00961 might be involved in multiple cancer-associated pathways such as chemokine, Ras and PI3K-Akt signaling pathways, GPCR ligand binding, and signal transduction in luminal subtypes of BC.	Phenobarbital	204-211	AKT serine/threonine kinase 1	Homo sapiens	132-135