PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32344356-0	2020	Cafestol preconditioning attenuates apoptosis and autophagy during hepatic ischemia-reperfusion injury by inhibiting ERK/PPARgamma pathway.	cafestol	0-8	peroxisome proliferator activated receptor gamma	Mus musculus	121-130	
32344356-4	2020	RESULTS: Our findings confirmed that cafestol preconditioning groups could reduce the levels of ALT and AST, alleviate liver pathological damage, suppress the release of inflammation mediators, inhibit the production of pro-apoptosis protein including caspase-3, caspase-9 and Bax, decrease the expression of autophagy-linked protein including Beclin-1 and LC3, increase anti-apoptosis protein Bcl-2, and restrain the activation of ERK and PPARgamma.	cafestol	37-45	peroxisome proliferator activated receptor gamma	Mus musculus	440-449	
32344356-5	2020	CONCLUSION: Cafestol preconditioning could attenuate inflammatory response, apoptosis and autophagy on hepatic ischemia reperfusion injury by suppressing ERK/PPARgamma pathway.	cafestol	12-20	peroxisome proliferator activated receptor gamma	Mus musculus	158-167