PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 28834568-7 2018 The aryl hydrocarbon receptor antagonist/CYP1A1 inhibitor alpha-naphthoflavone decreased luciferase induction and EROD activity by several compounds, including the methylated chrysenes, suggesting that metabolites of these chemicals contributed to ER activation. alpha-naphthoflavone 58-78 estrogen receptor 1 Homo sapiens 114-116 16488130-9 2006 The addition of other AhR ligands, alpha-naphthoflavone (alpha-NF, 10 microM) and luteolin (10 microM), to the culture media resulted in a similar suppression in ER-alpha mRNA levels to that caused by 5 microM DIM. alpha-naphthoflavone 35-55 estrogen receptor 1 Homo sapiens 162-170 16488130-9 2006 The addition of other AhR ligands, alpha-naphthoflavone (alpha-NF, 10 microM) and luteolin (10 microM), to the culture media resulted in a similar suppression in ER-alpha mRNA levels to that caused by 5 microM DIM. alpha-naphthoflavone 57-65 estrogen receptor 1 Homo sapiens 162-170 8276131-3 1993 Time-course studies showed that the decreases in nuclear ER and ER-ERE binding in TCDD-treated cells were observed within 1 to 3 h after treatment, respectively, and persisted for up to 24 h. Cycloheximide (10 microM) did not affect the TCDD-mediated response, whereas 1 microM alpha-naphthoflavone, an aryl hydrocarbon (Ah) receptor antagonist, partially blocked downregulation of nuclear ER binding by TCDD. alpha-naphthoflavone 278-298 estrogen receptor 1 Homo sapiens 57-59 8276131-3 1993 Time-course studies showed that the decreases in nuclear ER and ER-ERE binding in TCDD-treated cells were observed within 1 to 3 h after treatment, respectively, and persisted for up to 24 h. Cycloheximide (10 microM) did not affect the TCDD-mediated response, whereas 1 microM alpha-naphthoflavone, an aryl hydrocarbon (Ah) receptor antagonist, partially blocked downregulation of nuclear ER binding by TCDD. alpha-naphthoflavone 278-298 estrogen receptor 1 Homo sapiens 64-66 8276131-3 1993 Time-course studies showed that the decreases in nuclear ER and ER-ERE binding in TCDD-treated cells were observed within 1 to 3 h after treatment, respectively, and persisted for up to 24 h. Cycloheximide (10 microM) did not affect the TCDD-mediated response, whereas 1 microM alpha-naphthoflavone, an aryl hydrocarbon (Ah) receptor antagonist, partially blocked downregulation of nuclear ER binding by TCDD. alpha-naphthoflavone 278-298 estrogen receptor 1 Homo sapiens 64-66