PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
16684659-1	2006	Trace metals and polycyclic aromatic hydrocarbons (PAHs) are ubiquitous environmental co-contaminants and the trace metals could influence the carcinogenicity of the PAHs by altering their extent of induction of cytochromes P4501A1, 1A2, and 1B1 (CYP).	Polycyclic Aromatic Hydrocarbons	17-49	peptidylprolyl isomerase G	Homo sapiens	247-250	
17060372-10	2007	Overall, these data suggest that components within complex mixtures can alter PAH-induced carcinogenesis by inhibiting CYP bioactivation and influence other genotoxic effects, such as oxidative DNA damage.	Polycyclic Aromatic Hydrocarbons	78-81	peptidylprolyl isomerase G	Homo sapiens	119-122	
19568886-4	2008	Here, we have reviewed various aspects of CYPs for biomonitoring environmental health in terms of the CYP substrates, such as PAHs, aromatic amines, benzene/toluene, and tobacco smoking-related nitrosamines.	Polycyclic Aromatic Hydrocarbons	126-130	peptidylprolyl isomerase G	Homo sapiens	42-45	
16684659-1	2006	Trace metals and polycyclic aromatic hydrocarbons (PAHs) are ubiquitous environmental co-contaminants and the trace metals could influence the carcinogenicity of the PAHs by altering their extent of induction of cytochromes P4501A1, 1A2, and 1B1 (CYP).	Polycyclic Aromatic Hydrocarbons	51-55	peptidylprolyl isomerase G	Homo sapiens	247-250