PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
15671210-1	2005	We hypothesized that the risk of colorectal cancer associated with meat preparation methods producing heterocyclic amines or polycyclic aromatic hydrocarbons is modified by the CYP1A1 genotype alone or in combination with the GSTM1 genotype or the NAT2 imputed phenotype.	Polycyclic Aromatic Hydrocarbons	125-157	N-acetyltransferase 2	Homo sapiens	248-252	
12552997-1	2002	N-acetyltransferase-2 (NAT-2) and Glutathione-S-transferase M1 and T1 (GSTM1 and GSTT1) polymorphism have been implicated in the detoxification of urothelial carcinogens, such as arylamines and polycyclic aromatic hydrocarbons.	Polycyclic Aromatic Hydrocarbons	194-226	N-acetyltransferase 2	Homo sapiens	0-21	
23899393-0	1998	Influence of GSTM1 and NAT2 genotypes on the relationship between personal exposure to PAH and biomarkers of internal dose.	Polycyclic Aromatic Hydrocarbons	87-90	N-acetyltransferase 2	Homo sapiens	23-27	
23899393-4	1998	After stratifying by genetic polymorphism the correlation between personal PAH exposure and urinary PAH metabolites increased for individuals with NAT2 slow acetylators (r 0.58, p 0.001, n 29) and the combination of GSTM1 null and NAT2 slow acetylators (r 0.60, p 0.01, n 16).	Polycyclic Aromatic Hydrocarbons	75-78	N-acetyltransferase 2	Homo sapiens	147-151	
23899393-4	1998	After stratifying by genetic polymorphism the correlation between personal PAH exposure and urinary PAH metabolites increased for individuals with NAT2 slow acetylators (r 0.58, p 0.001, n 29) and the combination of GSTM1 null and NAT2 slow acetylators (r 0.60, p 0.01, n 16).	Polycyclic Aromatic Hydrocarbons	75-78	N-acetyltransferase 2	Homo sapiens	231-235	
23899393-4	1998	After stratifying by genetic polymorphism the correlation between personal PAH exposure and urinary PAH metabolites increased for individuals with NAT2 slow acetylators (r 0.58, p 0.001, n 29) and the combination of GSTM1 null and NAT2 slow acetylators (r 0.60, p 0.01, n 16).	Polycyclic Aromatic Hydrocarbons	100-103	N-acetyltransferase 2	Homo sapiens	147-151	
23899393-4	1998	After stratifying by genetic polymorphism the correlation between personal PAH exposure and urinary PAH metabolites increased for individuals with NAT2 slow acetylators (r 0.58, p 0.001, n 29) and the combination of GSTM1 null and NAT2 slow acetylators (r 0.60, p 0.01, n 16).	Polycyclic Aromatic Hydrocarbons	100-103	N-acetyltransferase 2	Homo sapiens	231-235	
8423501-4	1993	Cytochrome P4501A1, P4502E1 and N-acetyl transferase 2 are examples of enzymes involved in the metabolic activation of potential environmental carcinogens such as polycyclic aromatic hydrocarbons, benzene, and aromatic amines, respectively.	Polycyclic Aromatic Hydrocarbons	163-195	N-acetyltransferase 2	Homo sapiens	20-54	
31254350-2	2019	The present study was designed to investigate whether maternal NAT2 genetic polymorphisms are associated with fetal susceptibility to congenital heart diseases (CHDs) and to assess whether the risk is modified by polycyclic aromatic hydrocarbons (PAHs) exposure.	Polycyclic Aromatic Hydrocarbons	247-251	N-acetyltransferase 2	Homo sapiens	63-67	
28403014-6	2018	NAT2 slow genotype carriers had an OR of 3.59 (95% CI: 2.62-4.93) for BC when exposed to aromatic amines and an OR of 2.07 (95% CI: 1.36-3.15) when exposed to PAHs.	Polycyclic Aromatic Hydrocarbons	159-163	N-acetyltransferase 2	Homo sapiens	0-4	
28403014-8	2018	This meta-analysis indicates a possible association between the variant genotypes of GSTM1, GSTT1, NAT2 and SULT1A1, occupational exposure to aromatic amines or PAHs, and development of BC.	Polycyclic Aromatic Hydrocarbons	161-165	N-acetyltransferase 2	Homo sapiens	99-103