PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
9720771-2	1998	The purpose of the present study was to test the following hypothesis: propylthiouracil (PTU) treatments of rats induces an increase in the concentration and activity of the mitochondrial ATPase (m-ATPase) inhibitor protein (IF1).	Propylthiouracil	71-87	ATP synthase inhibitory factor subunit 1	Rattus norvegicus	225-228	
9720771-2	1998	The purpose of the present study was to test the following hypothesis: propylthiouracil (PTU) treatments of rats induces an increase in the concentration and activity of the mitochondrial ATPase (m-ATPase) inhibitor protein (IF1).	Propylthiouracil	89-92	ATP synthase inhibitory factor subunit 1	Rattus norvegicus	225-228	
9720771-8	1998	Propylthiouracil pretreatment for 12 days enhanced the concentration of the following metabolites in the liver: ATP (1.5 fold), ATPase inhibitor protein (IF1) (4.5 fold), and reduced glutathione (1.3 fold), while the activity of the inhibitor protein increased 2 fold.	Propylthiouracil	0-16	ATP synthase inhibitory factor subunit 1	Rattus norvegicus	154-157