PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
11745456-12	2001	These findings suggest that both ERK and cellular protein tyrosine kinase activation are involved in 12(S)-HETE-induced pancreatic cancer cell proliferation but P38 and JNK/SAPK are not involved in this mitogenic effect.	Hydroxyeicosatetraenoic Acids	107-111	mitogen-activated protein kinase 1	Homo sapiens	33-36	
9884018-2	1998	Oxidants activate cytosolic and membrane-bound PLA2 activities with the subsequent production of AA metabolites such as HETEs, which subsequently stimulate ERK and JNK (SAPK) activities leading to the activation of transcriptional factors and the ultimate stimulation of the transcription of several mitogen-stress-responsive genes.	Hydroxyeicosatetraenoic Acids	120-125	mitogen-activated protein kinase 1	Homo sapiens	156-159