PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 8538170-0 1995 Mechanisms of reoxygenation-induced calcium overload in cardiac myocytes: dependence on pHi. Calcium 36-43 glucose-6-phosphate isomerase 1 Mus musculus 88-91 8328315-1 1993 We found that the cytoplasmic concentration of calcium (Cai) of MC3T3-E1 osteoblasts was influenced by the type of pH buffer we used in the perfusing medium, suggesting that intracellular pH (pHi) might influence Cai. Calcium 47-54 glucose-6-phosphate isomerase 1 Mus musculus 192-195 2126845-0 1990 Effect of changes of pHi on intracellular calcium in a smooth muscle-like cell line. Calcium 42-49 glucose-6-phosphate isomerase 1 Mus musculus 21-24 30890468-4 2019 However, when extracellular calcium was removed, an instant depolarization and recovery of the pHi, back to normal levels, took place. Calcium 28-35 glucose-6-phosphate isomerase 1 Mus musculus 95-98 30890468-6 2019 Immunostaining and PCR data, showed that NBCe1 (SLC4A4) and NBCn1 (SLC4A7) were expressed in the cell population and that the pHi recovery in the radial glial-like cells after calcium removal was mediated mainly by the electrogenic sodium bicarbonate transporter NBCe1 (SLC4A4). Calcium 176-183 glucose-6-phosphate isomerase 1 Mus musculus 126-129 30890468-7 2019 Our results indicate that extracellular calcium might hamper pHi regulation and Na/HCO3 cotransporter activity in a brain injury microenvironment. Calcium 40-47 glucose-6-phosphate isomerase 1 Mus musculus 61-64 30890468-8 2019 Our findings show that the NBC-type transporters are the main pHi regulating systems prevailing in glia-like progenitor cells and that these calcium sensitive transporters are important for neuronal progenitor cell proliferation, survival and neural stem cell differentiation. Calcium 141-148 glucose-6-phosphate isomerase 1 Mus musculus 62-65 28409826-11 2018 GPi-s also activated the pathway of insulin secretion, indicated by enhanced glycolysis, mitochondrial oxidation and calcium signalling. Calcium 117-124 glucose-6-phosphate isomerase 1 Mus musculus 0-3 6338733-5 1983 The hypothesis that the calcium-activated potassium permeability is proton sensitive at an intracellular site, a fall in pHi causing a fall in permeability and an increase in pHi causing an increase in permeability, has been used to explain many of the effects observed in this study. Calcium 24-31 glucose-6-phosphate isomerase 1 Mus musculus 121-124 6338733-5 1983 The hypothesis that the calcium-activated potassium permeability is proton sensitive at an intracellular site, a fall in pHi causing a fall in permeability and an increase in pHi causing an increase in permeability, has been used to explain many of the effects observed in this study. Calcium 24-31 glucose-6-phosphate isomerase 1 Mus musculus 175-178