PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31888431-1	2020	Purpose: This study aimed to define the behavioral determinants influencing the decision of intensivists to consult a poison center (PC) when managing patients with calcium channel blocker (CCB) poisoning.Material and methods: Semi-structured interviews were conducted involving a convenience sample of 18 intensivists.	Calcium	165-172	pyruvate carboxylase	Homo sapiens	133-135	
19526286-9	2009	PCB 126 reduces NMDA-induced increase in calcium while PCB 153 does not.	Calcium	41-48	pyruvate carboxylase	Homo sapiens	0-3	
19429257-3	2009	We have previously shown that PCB activates human neutrophil granulocytes through a calcium dependent activation of phospholipase D and/or phospholipase C, followed by the activation of protein kinase C. In the present study, pharmacological characterization of Aroclor (A) 1242-induced respiratory burst in human neutrophils was conducted by the use of enzymatic inhibitors.	Calcium	84-91	pyruvate carboxylase	Homo sapiens	30-33	
19091378-1	2009	Treatment of PCBs in soil using metallic calcium and alcohol ultimately reduced 1300 x 10(-3)mgkg(-1) PCB concentration to 1.8 x 10(-3)mg kg(-1).	Calcium	41-48	pyruvate carboxylase	Homo sapiens	13-16	
9339820-15	1997	Additional research on the PCBs seems warranted in a number of areas, including the: 1) structural requirements necessary for binding to the Ah-receptor, 2) mechanism(s) of PCB-induced alterations in DA content and calcium homeostasis in vitro, 3) relationship between observed neurochemical effects in vitro and effects in vivo, and 4) relationship between PCB-induced neurochemical effects and crucial developmental processes such as those controlled by thyroid hormone development.	Calcium	215-222	pyruvate carboxylase	Homo sapiens	27-30	
9145907-0	1997	Noncoplanar PCB 95 alters microsomal calcium transport by an immunophilin FKBP12-dependent mechanism.	Calcium	37-44	pyruvate carboxylase	Homo sapiens	12-15	
9339820-15	1997	Additional research on the PCBs seems warranted in a number of areas, including the: 1) structural requirements necessary for binding to the Ah-receptor, 2) mechanism(s) of PCB-induced alterations in DA content and calcium homeostasis in vitro, 3) relationship between observed neurochemical effects in vitro and effects in vivo, and 4) relationship between PCB-induced neurochemical effects and crucial developmental processes such as those controlled by thyroid hormone development.	Calcium	215-222	pyruvate carboxylase	Homo sapiens	173-176	
6235862-8	1984	It is suggested that ATP-dependent binding of PCB- to SR membranes reflects calcium incorporation into the hydrophobic region of Ca2+-ATPase molecules.	Calcium	76-83	pyruvate carboxylase	Homo sapiens	46-49	
3921362-4	1985	We suggest that there may be a possible alteration in calcium metabolism in these babies, related to the fragile egg shells observed in PCB-contaminated birds and to the female hormone-enhancing effect of PCB.	Calcium	54-61	pyruvate carboxylase	Homo sapiens	136-139	
3921362-4	1985	We suggest that there may be a possible alteration in calcium metabolism in these babies, related to the fragile egg shells observed in PCB-contaminated birds and to the female hormone-enhancing effect of PCB.	Calcium	54-61	pyruvate carboxylase	Homo sapiens	205-208