PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24054501-10	2013	Proliferation of Huh 7/NTG cells was also inhibited by GCV treatment.	Ganciclovir	55-58	MIR7-3 host gene	Homo sapiens	17-22	
25545853-6	2015	In vivo therapeutic effects of AdAFP-TK and GCV in the HuH-7 xenograft model were monitored by (18)F-FDG PET.	Ganciclovir	44-47	MIR7-3 host gene	Homo sapiens	55-60	
19819065-4	2010	Our results show that: (1) all three genes are stably expressed in Huh-7/NTG cells, (2) I-125 and H3-PCV uptake were markedly increased in the Huh-7/NTG cells in vitro, (3) cellular survival and tumor growth of Huh-7/NTG was inhibited by I-131 or GCV both in vitro and in vivo, and was much prominent with combination therapy, (4) in vivo imaging with I-124 and F-18 FHBG revealed increased uptake in the Huh-7/NTG tumor.	Ganciclovir	247-250	MIR7-3 host gene	Homo sapiens	67-72	
19819065-4	2010	Our results show that: (1) all three genes are stably expressed in Huh-7/NTG cells, (2) I-125 and H3-PCV uptake were markedly increased in the Huh-7/NTG cells in vitro, (3) cellular survival and tumor growth of Huh-7/NTG was inhibited by I-131 or GCV both in vitro and in vivo, and was much prominent with combination therapy, (4) in vivo imaging with I-124 and F-18 FHBG revealed increased uptake in the Huh-7/NTG tumor.	Ganciclovir	247-250	MIR7-3 host gene	Homo sapiens	143-148	
19819065-4	2010	Our results show that: (1) all three genes are stably expressed in Huh-7/NTG cells, (2) I-125 and H3-PCV uptake were markedly increased in the Huh-7/NTG cells in vitro, (3) cellular survival and tumor growth of Huh-7/NTG was inhibited by I-131 or GCV both in vitro and in vivo, and was much prominent with combination therapy, (4) in vivo imaging with I-124 and F-18 FHBG revealed increased uptake in the Huh-7/NTG tumor.	Ganciclovir	247-250	MIR7-3 host gene	Homo sapiens	143-148	
19819065-4	2010	Our results show that: (1) all three genes are stably expressed in Huh-7/NTG cells, (2) I-125 and H3-PCV uptake were markedly increased in the Huh-7/NTG cells in vitro, (3) cellular survival and tumor growth of Huh-7/NTG was inhibited by I-131 or GCV both in vitro and in vivo, and was much prominent with combination therapy, (4) in vivo imaging with I-124 and F-18 FHBG revealed increased uptake in the Huh-7/NTG tumor.	Ganciclovir	247-250	MIR7-3 host gene	Homo sapiens	143-148	
11866443-4	2002	SCID mice of peritonitis carcinomatosis due to Huh-7 hepatoma cells infected with TOZ.1 could survive longer under administration of GCV than those without TOZ.1.	Ganciclovir	133-136	MIR7-3 host gene	Homo sapiens	47-52	
17182598-4	2007	Using an athymic nude mouse model (BALB/c-nu/nu), primary s.c. tumors (HuH7; human HCC cells) were completely eradicated by rAd followed by treatment with ganciclovir.	Ganciclovir	155-166	MIR7-3 host gene	Homo sapiens	71-75	
9852281-2	1998	GCV treatment resulted in pronounced growth inhibition of the virus-infected HuH-7 xenograft in mice, but did not affect growth of the parental xenograft.	Ganciclovir	0-3	MIR7-3 host gene	Homo sapiens	77-82	
11402306-6	2001	Repeated transfection of AFPTK1 followed by GCV treatment markedly suppressed growth of HUH7 tumors, and apoptosis of HUH7 cells was recognized in the tumor.	Ganciclovir	44-47	MIR7-3 host gene	Homo sapiens	88-92	
7585589-5	1995	As expected, only transduced HuH7 cells were killed by GCV treatment.	Ganciclovir	55-58	MIR7-3 host gene	Homo sapiens	29-33	
7585589-7	1995	All HuH7 tumors that were transduced with either Av1AFPTK1 or Av1TK1 completely regressed after GCV treatment.	Ganciclovir	96-99	MIR7-3 host gene	Homo sapiens	4-8