PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
1389231-6	1992	However, protein kinase C activators such as prostaglandin F2 alpha, phorbol esters and diacylglycerol were able to mimic GH in promoting a maximal mitogenic-adipogenic response, indicating that the ability of GH to induce diacylglycerol production (Doglio et al., 1989; Catalioto et al., 1990) plays a prominent role in this process.	Diglycerides	88-102	growth hormone	Mus musculus	122-124	
8123005-3	1994	Since GH stimulates the formation of diacylglycerol from phosphatidylcholine independently of Ca2+ mobilization, the role of Ca2+ was studied in regard to LPL gene expression stimulated by GH.	Diglycerides	37-51	growth hormone	Mus musculus	6-8	
8462467-0	1993	The stimulatory effect of insulin on diacylglycerol generation in adipocyte membranes from ob/ob mice is impaired by growth hormone.	Diglycerides	37-51	growth hormone	Mus musculus	117-131	
1389231-6	1992	However, protein kinase C activators such as prostaglandin F2 alpha, phorbol esters and diacylglycerol were able to mimic GH in promoting a maximal mitogenic-adipogenic response, indicating that the ability of GH to induce diacylglycerol production (Doglio et al., 1989; Catalioto et al., 1990) plays a prominent role in this process.	Diglycerides	88-102	growth hormone	Mus musculus	210-212	
1389231-6	1992	However, protein kinase C activators such as prostaglandin F2 alpha, phorbol esters and diacylglycerol were able to mimic GH in promoting a maximal mitogenic-adipogenic response, indicating that the ability of GH to induce diacylglycerol production (Doglio et al., 1989; Catalioto et al., 1990) plays a prominent role in this process.	Diglycerides	223-237	growth hormone	Mus musculus	122-124	
1389231-6	1992	However, protein kinase C activators such as prostaglandin F2 alpha, phorbol esters and diacylglycerol were able to mimic GH in promoting a maximal mitogenic-adipogenic response, indicating that the ability of GH to induce diacylglycerol production (Doglio et al., 1989; Catalioto et al., 1990) plays a prominent role in this process.	Diglycerides	223-237	growth hormone	Mus musculus	210-212	
2493151-6	1989	We conclude that the transduction signal of GH action in c-fos mRNA induction is the formation of diacylglycerol and that the mechanism whereby GH can activate protein kinase C is associated with a phospholipase C-mediated hydrolysis of glycerophospholipids other than inositol phospholipids.	Diglycerides	98-112	growth hormone	Mus musculus	44-46	
2125219-0	1990	Diacylglycerol production induced by growth hormone in Ob1771 preadipocytes arises from phosphatidylcholine breakdown.	Diglycerides	0-14	growth hormone	Mus musculus	37-51	
2125219-1	1990	Growth Hormone has recently been shown to stimulate the formation of diacylglycerol in Ob1771 mouse preadipocyte cells without increasing inositol lipid turnover.	Diglycerides	69-83	growth hormone	Mus musculus	0-14	
2125219-2	1990	Addition of growth hormone to Ob1771 cells prelabelled with [3H]glycerol or [3H]choline led to a rapid, transient and stoechiometric formation of labelled diacylglycerol and phosphocholine, respectively.	Diglycerides	155-169	growth hormone	Mus musculus	12-26	
2125219-5	1990	Pertussis toxin abolished both diacylglycerol and phosphocholine formation induced by growth hormone.	Diglycerides	31-45	growth hormone	Mus musculus	86-100	
2125219-6	1990	It is concluded that growth hormone mediates diacylglycerol production in Ob1771 cells by means of phosphatidylcholine breakdown involving a phospholipase C which is likely coupled to the growth hormone receptor via a pertussis toxin-sensitive G-protein.	Diglycerides	45-59	growth hormone	Mus musculus	21-35