PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
20039738-5	2010	Even in the presence of the more toxic arsenic species, arsenite, cell metabolism was significantly impaired only at the highest arsenite concentration (500 muM) for one of the Fe(II)-oxidizers.	arsenite	56-64	latexin	Homo sapiens	157-160	
20039738-5	2010	Even in the presence of the more toxic arsenic species, arsenite, cell metabolism was significantly impaired only at the highest arsenite concentration (500 muM) for one of the Fe(II)-oxidizers.	arsenite	129-137	latexin	Homo sapiens	157-160	
29882401-7	2018	The results showed that the mutagenic effects of arsenite (10 muM) and silver nanoparticles (10 mug/mL) were 70 +- 3 and 99 +- 7 per 105 survivors, respectively.	arsenite	49-57	latexin	Homo sapiens	62-65	
28025110-7	2017	In vitro experiments confirmed that arsenite treatment at short-term high doses (10-100muM) and long-term low doses (0.5-1muM) in human lymphoblasts (RPMI 1788) caused promoter hypomethylation of these genes, which was in concordance with an increase in their mRNA expression.	arsenite	36-44	latexin	Homo sapiens	87-90	
26731009-6	2016	Compared to the only DOX-loaded vesicle, the DOX-As-co-loaded one induced more than twice the apoptotic ratio of MCF-7/ADR breast cancer cells at a low As concentration (0.5 muM), due to the synergistic effects of DOX and As.	arsenite	49-51	latexin	Homo sapiens	174-177	
22551203-6	2012	In the in vitro study, human lymphoblasts were treated with arsenite at 0-100 muM for two, four and eight hours (short-term) and at 0, 0.5 and 1.0 muM for eight-weeks period (long-term).	arsenite	60-68	latexin	Homo sapiens	78-81	
26864988-5	2016	This conceptual self-powered arsenic biosensor demonstrated limits of detection (LODs) of 13 muM for arsenite and 132 muM for arsenate.	arsenite	101-109	latexin	Homo sapiens	93-96	
26823637-4	2015	The results of shaken batch bioassays indicated that the original, unexposed sludge was severely inhibited by arsenite (AsIII) as evidenced by the low 50% inhibition concentrations (IC50) determined, i.e., 19 and 90 muM for acetoclastic- and hydrogenotrophic methanogenesis, respectively.	arsenite	110-118	latexin	Homo sapiens	216-219	
24912785-3	2015	The present research aimed to determine whether miRNAs secreted from human bronchial epithelial (HBE) cells transformed by 1.0 muM arsenite are transferred into normal HBE cells and are functionally active in the recipient cells.	arsenite	131-139	latexin	Homo sapiens	127-130	
22244921-3	2012	Pretreatment with U-73122 (a specific PLC inhibitor) or 2-APB (a specific IP3 receptor antagonist) attenuated this effect, suggesting that PLC/IP3 signaling cascade is involved in arsenite-induced elevation of [Ca2+]i. Cytotoxic concentrations of arsenite (5 and 10 muM) significantly enhanced endothelial nitric oxide synthase (eNOS) phosphorylation, nitric oxide (NO) production and apoptosis after 24-h exposure.	arsenite	180-188	latexin	Homo sapiens	266-269	
22244921-5	2012	Moreover, we also found that non-apoptotic concentrations of arsenite (0.5 and 1 muM) dramatically mitigated thrombin-induced rapid transient rise of [Ca2+]i, eNOS phosphorylation and NO production, suggesting functional disruption of endothelial by arsenite, and these effects occurred without an alteration of PLC-beta1 and thrombin receptor levels.	arsenite	61-69	latexin	Homo sapiens	81-84	
20196163-4	2011	The 2,4-dinitrophenylhydrazine (DNPH) dot-blot assay revealed that arsenite (0-50 muM) dose-dependently increased protein carbonylation.	arsenite	67-75	latexin	Homo sapiens	82-85