PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30962733-7	2019	Some types of ginsenosides, including Rh1, Rg3, Rb1, compound K, chikusetsu saponin IVa, Rg5, and Rg1, have been clearly demonstrated to inhibit inflammatory responses by suppressing the activation of various inflammasomes, including the NLRP3, NLRP1, and absent in melanoma 2 inflammasomes.	Ginsenosides	14-26	NLR family pyrin domain containing 3	Homo sapiens	238-243	
28261091-6	2017	Ginsenoside Rg5 treatment suppressed NLRP3 inflammasome activation, preserved PDE3B expression and then reduced cAMP accumulation, contributing to inhibition of lipolysis.	Ginsenosides	0-11	NLR family pyrin domain containing 3	Homo sapiens	37-42	
28261091-10	2017	These findings establish a previously unrecognized role of ginsenosides in the regulation of lipid and glucose homeostasis and suggest that adipose succinate-associated NLRP3 inflammasome activation might be targeted therapeutically to prevent lipolysis and insulin resistance.	Ginsenosides	59-71	NLR family pyrin domain containing 3	Homo sapiens	169-174