PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23412769-7	2013	Activation of Bak, but not Bax, resulting from DATS treatment was markedly suppressed by overexpression of Mn-SOD.	diallyl trisulfide	47-51	BCL2 antagonist/killer 1	Homo sapiens	14-17	
18800351-0	2009	Diallyl trisulfide selectively causes Bax- and Bak-mediated apoptosis in human lung cancer cells.	diallyl trisulfide	0-18	BCL2 antagonist/killer 1	Homo sapiens	47-50	
18800351-10	2009	In conclusion, the present study indicates that Bax and Bak proteins are critical targets of DATS-induced apoptosis in human lung cancer cells.	diallyl trisulfide	93-97	BCL2 antagonist/killer 1	Homo sapiens	56-59	
18800351-8	2009	Knockdown of Bax and Bak proteins conferred significant protection against DATS-induced apoptotic cytoplasmic histone-associated DNA fragmentation.	diallyl trisulfide	75-79	BCL2 antagonist/killer 1	Homo sapiens	21-24	
17513609-0	2007	Mitochondria-mediated apoptosis by diallyl trisulfide in human prostate cancer cells is associated with generation of reactive oxygen species and regulated by Bax/Bak.	diallyl trisulfide	35-53	BCL2 antagonist/killer 1	Homo sapiens	163-166	
17513609-8	2007	In conclusion, the present study reveals that the mitochondria-mediated cell death by DATS is associated with ROS generation and regulated by Bax/Bak but independent of Bcl-2 or Bcl-xL.	diallyl trisulfide	86-90	BCL2 antagonist/killer 1	Homo sapiens	146-149	
17121905-0	2006	Diallyl trisulfide suppresses growth of PC-3 human prostate cancer xenograft in vivo in association with Bax and Bak induction.	diallyl trisulfide	0-18	BCL2 antagonist/killer 1	Homo sapiens	113-116