PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33780033-9	2021	Vitamin K2 alone tended to suppress the secretion of IL-17, IFN-gamma and TNF-alpha from the activated PBMCs of RA patients with smaller influences on the cytokine productions from healthy PBMCs.	Vitamin K 2	0-10	tumor necrosis factor	Homo sapiens	74-83	
33390552-10	2021	Vitamin K2 significantly inhibited the IL-17A, IL-10, and tumor necrosis factor alpha (TNF-alpha) production (p < 0.05), and increased the IL-2 production (p < 0.01) in the culture supernatant of atopic dermatitis PBMCs.	Vitamin K 2	0-10	tumor necrosis factor	Homo sapiens	58-85	
33390552-10	2021	Vitamin K2 significantly inhibited the IL-17A, IL-10, and tumor necrosis factor alpha (TNF-alpha) production (p < 0.05), and increased the IL-2 production (p < 0.01) in the culture supernatant of atopic dermatitis PBMCs.	Vitamin K 2	0-10	tumor necrosis factor	Homo sapiens	87-96	
21072493-7	2011	Furthermore, vitamin K2 prevented repression by tumor necrosis factor alpha (TNFalpha) of SMAD signaling induced by either transforming growth factor ss (TGFss) or bone morphogenetic protein-2 (BMP-2).	Vitamin K 2	13-23	tumor necrosis factor	Homo sapiens	48-75	
21072493-7	2011	Furthermore, vitamin K2 prevented repression by tumor necrosis factor alpha (TNFalpha) of SMAD signaling induced by either transforming growth factor ss (TGFss) or bone morphogenetic protein-2 (BMP-2).	Vitamin K 2	13-23	tumor necrosis factor	Homo sapiens	77-85	
17404108-9	2007	Vitamin K2 also suppressed the NF-kappaB binding site-dependent cyclin D1 promoter activity and suppressed the basal, 12-O-tetradecanoylphorbol-13-acetate (TPA)-, TNF-alpha-, and interleukin (IL)-1-induced activation of NF-kappaB binding and transactivation.	Vitamin K 2	0-10	tumor necrosis factor	Homo sapiens	163-172