PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 27093577-8 2016 DNS-DOX, however, effectively killed GSTP1 (20 nmol/min/mg) and MGST1 (450 nmol/min/mg) overexpressing cells as did the less reactive 4-mononitrobenzenesulfonyl doxorubicin (MNS-DOX) in a MGST1-dependent manner (1.5 nmol/min/mg) as shown previously. dns-dox 0-7 microsomal glutathione S-transferase 1 Homo sapiens 64-69 27093577-8 2016 DNS-DOX, however, effectively killed GSTP1 (20 nmol/min/mg) and MGST1 (450 nmol/min/mg) overexpressing cells as did the less reactive 4-mononitrobenzenesulfonyl doxorubicin (MNS-DOX) in a MGST1-dependent manner (1.5 nmol/min/mg) as shown previously. dns-dox 0-7 microsomal glutathione S-transferase 1 Homo sapiens 188-193 21851097-6 2011 Here we synthesized two derivatives of DOX, 2,4-dinitrobenzenesulfonyl doxorubicin (DNS-DOX) and 4-mononitrobenzenesulfonyl doxorubicin (MNS-DOX) and showed that they are substrates for MGST1 and GSTP (releasing DOX). dns-dox 84-91 microsomal glutathione S-transferase 1 Homo sapiens 186-191