PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
29307859-8	2019	The expression levels of some key proteins, responsible for proper S(E)R-mitochondria coupling such as Mfn-1, Fis-1, OPA1, BAP31, STIM1 and PML in either HG- or DOX-cells were supported our above hypothesis, strongly.	Doxorubicin	161-164	PML nuclear body scaffold	Homo sapiens	140-143	
22869143-3	2013	Doxorubicin, a common chemotherapeutic agent, is known to promote PML-mediated p53 activation in part by promoting PML-dependent MDM2 nucleolar sequestration.	Doxorubicin	0-11	PML nuclear body scaffold	Homo sapiens	66-69	
22869143-3	2013	Doxorubicin, a common chemotherapeutic agent, is known to promote PML-mediated p53 activation in part by promoting PML-dependent MDM2 nucleolar sequestration.	Doxorubicin	0-11	PML nuclear body scaffold	Homo sapiens	115-118	
22869143-4	2013	We discovered that BMK1 deactivation coupled with doxorubicin synergistically enhanced MDM2 nucleolar sequestration and, consequently, promoted PML-mediated p53 upregulation leading to tumor cell apoptosis in vitro and tumor regression in vivo.	Doxorubicin	50-61	PML nuclear body scaffold	Homo sapiens	144-147	
29735542-9	2018	Knockout of PML abolished doxorubicin-promoted DNA modification.	Doxorubicin	26-37	PML nuclear body scaffold	Homo sapiens	12-15	
21057547-5	2011	Exposure to genotoxic signals including UV and doxorubicin induces AXIN to enter into the nucleus where it colocalizes with PML in the nuclear bodies.	Doxorubicin	47-58	PML nuclear body scaffold	Homo sapiens	124-127