PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25203837-1	2014	The aims of this study were to distinguish between the primary and secondary effects of TGF-beta signalling disruption by Dox treatment in NTPDase2+ cells; and to investigate the interactions between TGF-beta signalling and Jagged2/Notch1 pathway in regulating the expansion of tongue epithelia stem cells.Transgenic mice expressing rtTA from the mouse NTPDase2 promoter or K14 promoter were used to generate an inducible dominant negative TGF-beta receptor type II (Tgfbr2) mutant model.Disruption of TGF-beta signalling in NTPDase2+ cells initially inhibited the formation of filiform papillae but led to their regeneration over time.	Doxorubicin	122-125	ectonucleoside triphosphate diphosphohydrolase 2	Mus musculus	139-147