PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31887299-8	2020	Independent of mTOR -ULK1 signaling pathway, activation of autophagy of DNR was proved to be mediated by AMPK (pThr172)/ULK1 pathway.	Daunorubicin	72-75	unc-51 like autophagy activating kinase 1	Homo sapiens	21-25	
31887299-0	2020	Targeted inhibition of ULK1 enhances daunorubicin sensitivity in acute myeloid leukemia.	Daunorubicin	37-49	unc-51 like autophagy activating kinase 1	Homo sapiens	23-27	
31887299-7	2020	Furthermore, ULK1 inhibition by a highly selective kinase inhibitor SBI-0206965 and shRNA enhanced cytotoxicity of DNR against AML cells.	Daunorubicin	115-118	unc-51 like autophagy activating kinase 1	Homo sapiens	13-17	
31887299-8	2020	Independent of mTOR -ULK1 signaling pathway, activation of autophagy of DNR was proved to be mediated by AMPK (pThr172)/ULK1 pathway.	Daunorubicin	72-75	unc-51 like autophagy activating kinase 1	Homo sapiens	120-124	
31887299-9	2020	CONCLUSIONS: These results revealed that pro-survival autophagy induced by ULK1 activation was one of the potential mechanisms of AML resistance to DNR.	Daunorubicin	148-151	unc-51 like autophagy activating kinase 1	Homo sapiens	75-79	
31887299-10	2020	Targeting ULK1 selectively could be a promising therapeutic strategy to enhance sensitivity of DNR for AML therapy.	Daunorubicin	95-98	unc-51 like autophagy activating kinase 1	Homo sapiens	10-14