PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26118906-10	2015	Analysis of niclosamide"s effect on 11 cancer-related signal pathway reporters reveals that three of them, the E2F1, AP1, and c-Myc-responsive reporters, are significantly inhibited.	Niclosamide	12-23	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	117-120	
24750999-0	2014	Niclosamide enhances ROS-mediated cell death through c-Jun activation.	Niclosamide	0-11	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	53-58	
24750999-8	2014	Niclosamide pretreatment also induced c-Jun and its phosphorylation in the presence of H2O2, thereby enhancing apoptosis.	Niclosamide	0-11	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	38-43	
24750999-10	2014	Knockdown of c-Jun also decreased PARP cleavage and clonogenic cell survival in niclosamide- and IR-treated H1299 cells.	Niclosamide	80-91	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	13-18	
24750999-11	2014	Our findings suggest that niclosamide could be a promising radiosensitizer in lung cancer patients through activation of the p38 MAPK-c-Jun axis.	Niclosamide	26-37	Jun proto-oncogene, AP-1 transcription factor subunit	Homo sapiens	134-139