PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
28102849-6	2017	BBR-SDT also reduced the phosphorylation of Akt and mTOR, two key molecules in the PI3K/AKT/mTOR signaling pathway, which is responsible for inducing autophagy.	bbr-sdt	0-7	mechanistic target of rapamycin kinase	Homo sapiens	52-56	
28102849-6	2017	BBR-SDT also reduced the phosphorylation of Akt and mTOR, two key molecules in the PI3K/AKT/mTOR signaling pathway, which is responsible for inducing autophagy.	bbr-sdt	0-7	mechanistic target of rapamycin kinase	Homo sapiens	92-96	
28102849-9	2017	Taken together, these results demonstrate that BBR-SDT effectively promotes cholesterol efflux by increasing reactive oxygen species generation, and this subsequently induces autophagy via the PI3K/AKT/mTOR signaling pathway in both "normal" macrophages and lipid-loaded macrophages (foam cells).	bbr-sdt	47-54	mechanistic target of rapamycin kinase	Homo sapiens	202-206