PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 34400868-7 2021 Additionally, candesartan and/or CoQ10 diminished gastrocnemius active caspase-3 level and phospho-p38 MAPK protein expression. coenzyme Q10 33-38 caspase 3 Rattus norvegicus 71-80 30393023-10 2019 ALA and/or CoQ10 was proved to prevent apoptosis and degeneration of DRG neurons, which appears to be mediated by regulating the expression of caspase 3 and UCP2 proteins, inducing ATP and improving DN-induced changes in DRG neurons. coenzyme Q10 11-16 caspase 3 Rattus norvegicus 143-152 33368479-8 2021 In addition, a decrease in the expression of caspase3 and caspase 8 was viewed in coenzyme Q10-treated groups. coenzyme Q10 82-94 caspase 3 Rattus norvegicus 45-53 33393703-7 2021 Furthermore, EGCG + CoQ10 treatment inhibited CP-induced apoptosis by suppressing the activation and mitochondrial accumulation of proapoptotic proteins and preventing the inhibition of antiapoptotic protein expression, cytochrome c efflux, caspase-3 activation, and DNA fragmentation. coenzyme Q10 20-25 caspase 3 Rattus norvegicus 241-250 31737205-5 2019 Our results showed that CoQ10 treatment could significantly decrease the levels of oxidative products (MDA) and increase the activities of antioxidant enzymes (SOD and GSH) against oxidative stress, as well as decrease the levels of pro-apoptotic proteins (Bax and Caspase-3) and increase the levels of anti-apoptotic proteins (Bcl-2) against apoptosis after SCI. coenzyme Q10 24-29 caspase 3 Rattus norvegicus 265-274 34939895-12 2021 Co-administration of CoQ10 resulted in significant improvement in the histopathological picture, with a significant decrease in caspase-3 and iNOS immunoexpression and downregulation of the Bax/Bcl-2 gene expression ratio. coenzyme Q10 21-26 caspase 3 Rattus norvegicus 128-137