PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34939895-12	2021	Co-administration of CoQ10 resulted in significant improvement in the histopathological picture, with a significant decrease in caspase-3 and iNOS immunoexpression and downregulation of the Bax/Bcl-2 gene expression ratio.	coenzyme Q10	21-26	caspase 3	Rattus norvegicus	128-137	
33393703-7	2021	Furthermore, EGCG + CoQ10 treatment inhibited CP-induced apoptosis by suppressing the activation and mitochondrial accumulation of proapoptotic proteins and preventing the inhibition of antiapoptotic protein expression, cytochrome c efflux, caspase-3 activation, and DNA fragmentation.	coenzyme Q10	20-25	caspase 3	Rattus norvegicus	241-250	
33368479-8	2021	In addition, a decrease in the expression of caspase3 and caspase 8 was viewed in coenzyme Q10-treated groups.	coenzyme Q10	82-94	caspase 3	Rattus norvegicus	45-53	
31737205-5	2019	Our results showed that CoQ10 treatment could significantly decrease the levels of oxidative products (MDA) and increase the activities of antioxidant enzymes (SOD and GSH) against oxidative stress, as well as decrease the levels of pro-apoptotic proteins (Bax and Caspase-3) and increase the levels of anti-apoptotic proteins (Bcl-2) against apoptosis after SCI.	coenzyme Q10	24-29	caspase 3	Rattus norvegicus	265-274	
30393023-10	2019	ALA and/or CoQ10 was proved to prevent apoptosis and degeneration of DRG neurons, which appears to be mediated by regulating the expression of caspase 3 and UCP2 proteins, inducing ATP and improving DN-induced changes in DRG neurons.	coenzyme Q10	11-16	caspase 3	Rattus norvegicus	143-152	
34400868-7	2021	Additionally, candesartan and/or CoQ10 diminished gastrocnemius active caspase-3 level and phospho-p38 MAPK protein expression.	coenzyme Q10	33-38	caspase 3	Rattus norvegicus	71-80