PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24868010-7	2014	Despite the absence of exogenous aldosterone, these effects were prevented by the aldosterone synthase inhibitor FAD286 (100 nM) or the mineralocorticoid receptor (MR)-antagonist spironolactone (100 nM), indicating endogenous aldosterone synthesis and MR-dependent signaling.	Fadrozole	113-119	nuclear receptor subfamily 3, group C, member 2	Mus musculus	252-254	
24868010-8	2014	Interestingly, in the presence of high-sodium concentrations, FAD286 increased the transcription of the MR by 69%.	Fadrozole	62-68	nuclear receptor subfamily 3, group C, member 2	Mus musculus	104-106