PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
22188633-5	2012	Activation of mGluR5 with (RS)-3, 5-dihydroxy phenylglycine (DHPG) promoted cardiomyocyte differentiation in a dose-dependent manner.	dhpg	61-65	glutamate receptor, ionotropic, kainate 1	Mus musculus	14-20	
26902516-4	2016	Treatment with DHPG (100 muM) also promoted the proliferation of RPCs, as indicated by 5-Bromo-2-deoxyUridine (BrdU) staining and flow cytometry, and likewise, MTEP (100 muM) and mGluR5 knockdown abolished the action of mGluR5 activity.	dhpg	15-19	glutamate receptor, ionotropic, kainate 1	Mus musculus	179-185	
26902516-4	2016	Treatment with DHPG (100 muM) also promoted the proliferation of RPCs, as indicated by 5-Bromo-2-deoxyUridine (BrdU) staining and flow cytometry, and likewise, MTEP (100 muM) and mGluR5 knockdown abolished the action of mGluR5 activity.	dhpg	15-19	glutamate receptor, ionotropic, kainate 1	Mus musculus	220-226	
26902516-5	2016	Western blot demonstrated that the activation of mGluR5 enhanced the expression of Cyclin D1 and the phosphorylation level of PKC however, MTEP or mGluR5 knockdown also abrogated the effect of DHPG on RPCs.	dhpg	193-197	glutamate receptor, ionotropic, kainate 1	Mus musculus	49-55	
26902516-5	2016	Western blot demonstrated that the activation of mGluR5 enhanced the expression of Cyclin D1 and the phosphorylation level of PKC however, MTEP or mGluR5 knockdown also abrogated the effect of DHPG on RPCs.	dhpg	193-197	glutamate receptor, ionotropic, kainate 1	Mus musculus	147-153	
25421413-7	2015	The effects of DHPG were abolished by the mGluR1 antagonist LY367385 and the specific inhibitor of MEK1, PD98059 which also reduced phosphorylation of extracellular-signal-regulated protein kinase 1/2 (ERK1/2); but not by the mGluR5 antagonist 6-methyl-2-(phenylethynyl) pyridine hydrochloride or the selective inhibitor of protein kinase C (PKC).	dhpg	15-19	glutamate receptor, ionotropic, kainate 1	Mus musculus	226-232	
22188633-11	2012	mGluR5 siRNA also decreased the DHPG-induced Ca2+ transient peak amplitude in the isolated ESC-derived cardiomyocytes.	dhpg	32-36	glutamate receptor, ionotropic, kainate 1	Mus musculus	0-6	
21723923-3	2011	Results showed that mGluR5 agonist (S)-3,5-dihydroxyphenylglycine hydrate (DHPG) increased the proliferation of NPCs by increasing cell activity, diameter of neurospheres and cell division, while mGluR5 siRNA and antagonist 6-methyl-2-(phenylethynyl) pyridine hydrochloride (MPEP) decreased the NPC proliferation.	dhpg	75-79	glutamate receptor, ionotropic, kainate 1	Mus musculus	20-26	
21723923-3	2011	Results showed that mGluR5 agonist (S)-3,5-dihydroxyphenylglycine hydrate (DHPG) increased the proliferation of NPCs by increasing cell activity, diameter of neurospheres and cell division, while mGluR5 siRNA and antagonist 6-methyl-2-(phenylethynyl) pyridine hydrochloride (MPEP) decreased the NPC proliferation.	dhpg	75-79	glutamate receptor, ionotropic, kainate 1	Mus musculus	196-202