PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19641002-3	2009	In this study, we generated dominant Gag p11C- and subdominant Env p41A-specific CD8(+) T-lymphocyte responses in Mamu-A*01(+) rhesus monkeys through vaccination with plasmid DNA and recombinant adenovirus encoding simian-human immunodeficiency virus (SHIV) proteins.	Glycosaminoglycans	37-40	major histocompatibility complex, class I, A	Macaca mulatta	114-120	
19641002-4	2009	Infection of vaccinated Mamu-A*01(+) rhesus monkeys with a SHIV Gag Deltap11C mutant virus generated a significantly increased expansion of the Env p41A-specific CD8(+) T-lymphocyte response in the absence of secondary Gag p11C-specific CD8(+) T-lymphocyte responses.	Glycosaminoglycans	64-67	major histocompatibility complex, class I, A	Macaca mulatta	24-30	
8394161-3	1993	We have shown that SIVmac-infected rhesus monkeys expressing the major histocompatibility complex (MHC) class I molecule Mamu-A*01 develop a SIVmac gag-specific CTL response which recognizes a 9 amino acid fragment of the gag protein in association with Mamu-A*01.	Glycosaminoglycans	148-151	major histocompatibility complex, class I, A	Macaca mulatta	121-127	
8394161-3	1993	We have shown that SIVmac-infected rhesus monkeys expressing the major histocompatibility complex (MHC) class I molecule Mamu-A*01 develop a SIVmac gag-specific CTL response which recognizes a 9 amino acid fragment of the gag protein in association with Mamu-A*01.	Glycosaminoglycans	148-151	major histocompatibility complex, class I, A	Macaca mulatta	254-260	
1460291-2	1992	Previous studies have demonstrated that simian immunodeficiency virus of macaques (SIVmac)-infected rhesus monkeys expressing the MHC class I allele Mamu-A*01 reproducibly develop a gag-specific CTL response limited to a 9-amino acid epitope of the SIVmac gag protein (residues 182-190 within peptide 11C).	Glycosaminoglycans	182-185	major histocompatibility complex, class I, A	Macaca mulatta	149-155	
11752176-11	2002	After the acute phase, however, this pattern of reactivity changed, and the Mamu-A*01-restricted response against the Gag(181-189)CM9 epitope became dominant.	Glycosaminoglycans	118-121	major histocompatibility complex, class I, A	Macaca mulatta	76-82	
9637523-6	1998	We have now defined the optimal SIV gag CTL epitope restricted by the rhesus MHC class I molecule Mamu-A*01 and defined a general peptide binding motif for this molecule that is characterized by a dominant position 3 anchor (proline).	Glycosaminoglycans	36-39	major histocompatibility complex, class I, A	Macaca mulatta	98-104	
9637523-8	1998	Mamu-A*01 is unique in that it is found at a high frequency in rhesus macaques, and all SIV-infected Mamu-A*01-positive rhesus macaques studied to date develop an immunodominant gag-specific CTL response restricted by this molecule.	Glycosaminoglycans	178-181	major histocompatibility complex, class I, A	Macaca mulatta	0-6