PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33973360-0	2021	Rapid-acting antidepressants and the regulation of TrkB neurotrophic signalling - insights from ketamine, nitrous oxide, seizures, and anaesthesia.	Nitrous Oxide	106-119	neurotrophic receptor tyrosine kinase 2	Homo sapiens	51-55	
33973360-5	2021	We have shown nitrous oxide to evoke TrkB signalling preferentially after the acute pharmacological effects have dissipated (i.e. after receptor disengagement), when slow delta-frequency electroencephalogram (EEG) activity is upregulated.	Nitrous Oxide	14-27	neurotrophic receptor tyrosine kinase 2	Homo sapiens	37-41	
34464676-3	2021	Our recent studies suggest that a single subanesthetic dose of NMDAR antagonists ketamine or nitrous oxide (N2O) gradually evoke 1-4 Hz electrophysiological activity (delta-rhythm) of cerebral cortex that is accompanied by molecular signaling associated with synaptic plasticity (e.g. activation of tropomyosin receptor kinase B (TrkB) and inhibition of glycogen synthase kinase 3beta (GSK3beta)).	Nitrous Oxide	93-106	neurotrophic receptor tyrosine kinase 2	Homo sapiens	299-328	
34464676-3	2021	Our recent studies suggest that a single subanesthetic dose of NMDAR antagonists ketamine or nitrous oxide (N2O) gradually evoke 1-4 Hz electrophysiological activity (delta-rhythm) of cerebral cortex that is accompanied by molecular signaling associated with synaptic plasticity (e.g. activation of tropomyosin receptor kinase B (TrkB) and inhibition of glycogen synthase kinase 3beta (GSK3beta)).	Nitrous Oxide	93-106	neurotrophic receptor tyrosine kinase 2	Homo sapiens	330-334	
34464676-3	2021	Our recent studies suggest that a single subanesthetic dose of NMDAR antagonists ketamine or nitrous oxide (N2O) gradually evoke 1-4 Hz electrophysiological activity (delta-rhythm) of cerebral cortex that is accompanied by molecular signaling associated with synaptic plasticity (e.g. activation of tropomyosin receptor kinase B (TrkB) and inhibition of glycogen synthase kinase 3beta (GSK3beta)).	Nitrous Oxide	108-111	neurotrophic receptor tyrosine kinase 2	Homo sapiens	299-328	
34464676-3	2021	Our recent studies suggest that a single subanesthetic dose of NMDAR antagonists ketamine or nitrous oxide (N2O) gradually evoke 1-4 Hz electrophysiological activity (delta-rhythm) of cerebral cortex that is accompanied by molecular signaling associated with synaptic plasticity (e.g. activation of tropomyosin receptor kinase B (TrkB) and inhibition of glycogen synthase kinase 3beta (GSK3beta)).	Nitrous Oxide	108-111	neurotrophic receptor tyrosine kinase 2	Homo sapiens	330-334