PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
25043756-0	2014	Dihydrotestosterone enhances castration-resistant prostate cancer cell proliferation through STAT5 activation via glucocorticoid receptor pathway.	Dihydrotestosterone	0-19	nuclear receptor subfamily 3 group C member 1	Homo sapiens	114-137	
25043756-11	2014	On immunofluorescence, activation of STAT5 and GR translocating into the nucleus after DHT treatment was confirmed.	Dihydrotestosterone	87-90	nuclear receptor subfamily 3 group C member 1	Homo sapiens	47-49	
25043756-14	2014	Activation was induced regardless of presence of AR and in cells devoid of AR, DHT used GR which formed direct complex with p-STAT5.	Dihydrotestosterone	79-82	nuclear receptor subfamily 3 group C member 1	Homo sapiens	88-90	
16125883-3	2006	DHT induced AR-mediated transcriptional activity in a concentration-dependent manner with median effective concentration (EC(50)) value of 3.90 x 10(-10) M. Nilutamide exhibited potent antiandrogenic activity with median inhibitory concentration (IC(50)) value of 8.90 x 10(-7) M. The transcription could not be activated by glucocorticoid receptor (GR) agonist dexamethasone, which suggested that the assay system be highly specific to androgenic compounds without cross-talk to GR agonist.	Dihydrotestosterone	0-3	nuclear receptor subfamily 3 group C member 1	Homo sapiens	325-348	
16125883-3	2006	DHT induced AR-mediated transcriptional activity in a concentration-dependent manner with median effective concentration (EC(50)) value of 3.90 x 10(-10) M. Nilutamide exhibited potent antiandrogenic activity with median inhibitory concentration (IC(50)) value of 8.90 x 10(-7) M. The transcription could not be activated by glucocorticoid receptor (GR) agonist dexamethasone, which suggested that the assay system be highly specific to androgenic compounds without cross-talk to GR agonist.	Dihydrotestosterone	0-3	nuclear receptor subfamily 3 group C member 1	Homo sapiens	350-352	
16125883-3	2006	DHT induced AR-mediated transcriptional activity in a concentration-dependent manner with median effective concentration (EC(50)) value of 3.90 x 10(-10) M. Nilutamide exhibited potent antiandrogenic activity with median inhibitory concentration (IC(50)) value of 8.90 x 10(-7) M. The transcription could not be activated by glucocorticoid receptor (GR) agonist dexamethasone, which suggested that the assay system be highly specific to androgenic compounds without cross-talk to GR agonist.	Dihydrotestosterone	0-3	nuclear receptor subfamily 3 group C member 1	Homo sapiens	480-482	
28611094-5	2017	We used proximity-dependent biotin identification to map the protein interaction landscapes of GR and AR in the presence and absence of their cognate agonist (dexamethasone, 5alpha-dihydrotestosterone) and antagonist (RU486, enzalutamide) in intact human cells.	Dihydrotestosterone	174-200	nuclear receptor subfamily 3 group C member 1	Homo sapiens	95-97