PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
19296982-2	2009	Primary hyperoxaluria type 2 is a rare autosomal recessive disease caused by mutations in the GRHPR gene, leading to an accumulation of oxalate and L-glycerate with recurrent kidney stone formation and nephrocalcinosis, and the later development of renal failure and systemic oxalate depositions.	glyceric acid	148-159	glyoxylate and hydroxypyruvate reductase	Homo sapiens	94-99	
11156703-1	2000	Primary hyperoxaluria Type II (PH2) is a rare monogenic disease characterized by excessive urinary oxalate and L-glycerate excretion.	glyceric acid	111-122	glyoxylate and hydroxypyruvate reductase	Homo sapiens	31-34	
3036109-7	1987	FAB mass spectrometry, CID/MIKE spectra and a specific enzymatic assay with glyoxylate reductase, demonstrated that the 82 adduct was a glycerate moiety.	glyceric acid	136-145	glyoxylate and hydroxypyruvate reductase	Homo sapiens	76-96	
28681512-6	2018	Although requiring confirmation in additional cases, the normalization of plasma oxalate, urine oxalate, and urine glycerate levels observed in this patient within a month of the transplant that remain reduced at the most recent follow-up at 13 months suggests that correction of the GRHPR deficiency in PH2 can be achieved by liver transplantation.	glyceric acid	115-124	glyoxylate and hydroxypyruvate reductase	Homo sapiens	284-289	
12185464-2	2002	There are only 24 reported cases of PH2, which is characterized by raised urine oxalate and L-glycerate.	glyceric acid	92-103	glyoxylate and hydroxypyruvate reductase	Homo sapiens	36-39