PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10715809-7	2000	However, the vascular relaxation caused by insulin and IGF-I was completely abolished by KCI, and was attenuated by the nonspecific potassium-channel blocker tetraethylammonium (for IMA rings, to 77 +/- 8 and 66 +/- 4% with insulin and IGF-I, respectively; for saphenous vein rings, 73 +/- 2 and 77 +/- 1% for insulin and IGF-I, respectively, P < 0.001).	Tetraethylammonium	158-176	insulin like growth factor 1	Homo sapiens	55-60	
10715809-7	2000	However, the vascular relaxation caused by insulin and IGF-I was completely abolished by KCI, and was attenuated by the nonspecific potassium-channel blocker tetraethylammonium (for IMA rings, to 77 +/- 8 and 66 +/- 4% with insulin and IGF-I, respectively; for saphenous vein rings, 73 +/- 2 and 77 +/- 1% for insulin and IGF-I, respectively, P < 0.001).	Tetraethylammonium	158-176	insulin like growth factor 1	Homo sapiens	236-241	
10715809-7	2000	However, the vascular relaxation caused by insulin and IGF-I was completely abolished by KCI, and was attenuated by the nonspecific potassium-channel blocker tetraethylammonium (for IMA rings, to 77 +/- 8 and 66 +/- 4% with insulin and IGF-I, respectively; for saphenous vein rings, 73 +/- 2 and 77 +/- 1% for insulin and IGF-I, respectively, P < 0.001).	Tetraethylammonium	158-176	insulin like growth factor 1	Homo sapiens	236-241	
9719047-11	1998	KCl and TEA attenuated the vasorelaxation effect of both insulin and IGF-I.	Tetraethylammonium	8-11	insulin like growth factor 1	Homo sapiens	69-74