PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 19961834-0 2010 Group I metabotropic glutamate receptors are involved in TEA-induced long-term potentiation at mossy fiber-CA3 synapses in the rat hippocampus. Tetraethylammonium 57-60 carbonic anhydrase 3 Rattus norvegicus 107-110 19961834-2 2010 We electrophysiologically examined the involvement of the Group I mGluR in tetraethylammonium (TEA)-induced long-term potentiation (LTP) at mossy fiber (MF)-CA3 synapses in the rat hippocampus. Tetraethylammonium 75-93 carbonic anhydrase 3 Rattus norvegicus 157-160 19961834-2 2010 We electrophysiologically examined the involvement of the Group I mGluR in tetraethylammonium (TEA)-induced long-term potentiation (LTP) at mossy fiber (MF)-CA3 synapses in the rat hippocampus. Tetraethylammonium 95-98 carbonic anhydrase 3 Rattus norvegicus 157-160 19961834-6 2010 These results demonstrate the crucial roles of Group I mGluR in the TEA-induced LTP at MF-CA3 synapses and the different contributions of mGluR1 and mGluR5 to the initial component of plasticity. Tetraethylammonium 68-71 carbonic anhydrase 3 Rattus norvegicus 90-93 11535660-11 2001 The maximal slope of the Ca(2+)-dependent action potential of the CA3 neurons, which were evoked in the presence of 8-CPT (1 microM), nifedipine (20 microM), TTX (0.5 microM), and tetraethyl ammonium chloride (20 mM), decreased by 12 +/- 6% of the control 4 min after the onset of in vitro ischemia. Tetraethylammonium 180-208 carbonic anhydrase 3 Rattus norvegicus 66-69 18977337-0 2009 TEA-induced long-term potentiation at hippocampal mossy fiber-CA3 synapses: characteristics of its induction and expression. Tetraethylammonium 0-3 carbonic anhydrase 3 Rattus norvegicus 62-65 18977337-1 2009 Potassium ion channel blockade by tetraethylammonium (TEA) reportedly induces long-term potentiation (LTP) at hippocampal mossy fiber (MF)-CA3 synapses, but the characteristics of induction, expression, and modulation of the LTP remain unclear. Tetraethylammonium 34-52 carbonic anhydrase 3 Rattus norvegicus 139-142 18977337-1 2009 Potassium ion channel blockade by tetraethylammonium (TEA) reportedly induces long-term potentiation (LTP) at hippocampal mossy fiber (MF)-CA3 synapses, but the characteristics of induction, expression, and modulation of the LTP remain unclear. Tetraethylammonium 54-57 carbonic anhydrase 3 Rattus norvegicus 139-142 18977337-2 2009 In the present study, these features of TEA-induced LTP at MF-CA3 synapses were electrophysiologically examined using rat hippocampal slices. Tetraethylammonium 40-43 carbonic anhydrase 3 Rattus norvegicus 62-65 18977337-3 2009 Synaptic responses recorded from MF-CA3 synapses were enhanced long-term by TEA application even under the blockade of NMDA receptors with D-AP5, whereas selective pharmacological blockade of T-type voltage-dependent calcium channels (VDCCs) strongly inhibited TEA-induced LTP. Tetraethylammonium 76-79 carbonic anhydrase 3 Rattus norvegicus 36-39 18977337-5 2009 The facilitative modulation of MF-CA3 LTP by GABA(A) receptor activation reported previously was reversed when bumetanide, a blocker of Na(+)-K(+)-Cl(-) co-transporters (NKCCs), was applied, suggesting that the region-specific modulation of TEA-induced LTP by GABAergic inputs at MF-CA3 synapses is due to the dominance of NKCC action at MF terminals. Tetraethylammonium 241-244 carbonic anhydrase 3 Rattus norvegicus 34-37 17669533-0 2007 Regional differences in GABAergic modulation for TEA-induced synaptic plasticity in rat hippocampal CA1, CA3 and dentate gyrus. Tetraethylammonium 49-52 carbonic anhydrase 3 Rattus norvegicus 105-108 17669533-1 2007 Tetraethylammonium (TEA), a K(+)-channel blocker, reportedly induces long-term potentiation (LTP) of hippocampal CA1 synaptic responses, but at CA3 and the dentate gyrus (DG), the characteristics of TEA-induced plasticity and modulation by inhibitory interneurons remain unclear. Tetraethylammonium 0-18 carbonic anhydrase 3 Rattus norvegicus 144-147 17669533-1 2007 Tetraethylammonium (TEA), a K(+)-channel blocker, reportedly induces long-term potentiation (LTP) of hippocampal CA1 synaptic responses, but at CA3 and the dentate gyrus (DG), the characteristics of TEA-induced plasticity and modulation by inhibitory interneurons remain unclear. Tetraethylammonium 20-23 carbonic anhydrase 3 Rattus norvegicus 144-147 17669533-3 2007 In Schaffer collateral-CA1 synapses and associational fiber (AF)-CA3 synapses, bath application of TEA-induced LTP in the presence and absence of picrotoxin (PTX), a GABA(A) receptor blocker, whereas TEA-induced LTP at mossy fiber (MF)-CA3 synapses was detected only in the absence of GABA(A) receptor blockers. Tetraethylammonium 99-102 carbonic anhydrase 3 Rattus norvegicus 236-239 17669533-7 2007 This series of results demonstrates that TEA-induced DG and CA3 plasticity displays afferent specificity and is exposed to GABAergic modulation in an opposite manner. Tetraethylammonium 41-44 carbonic anhydrase 3 Rattus norvegicus 60-63 11726243-12 2001 Tetraethylammonium application also was associated with a large, late EPSP component in CA1 that persisted even after severing the connections between CA3 and CA1. Tetraethylammonium 0-18 carbonic anhydrase 3 Rattus norvegicus 151-154 7589175-5 1995 In the CA3 pyramidal neurons of SER, the Ca2+ spikes induced by the depolarizing pulse applied in the cell in the presence of tetrodotoxin and tetraethylammonium had a different configuration from that in normal Wistar rats. Tetraethylammonium 143-161 carbonic anhydrase 3 Rattus norvegicus 7-10