PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23874797-7	2013	Diabetes-induced abnormalities (leukostasis, ICAM-1 expression on the luminal surface of the vascular endothelium, retinal superoxide generation) were significantly inhibited by removing either MyD88 or the signaling pathways regulated by it (TLRs 2 and 4, and IL-1ss) from bone marrow-derived cells only.	Superoxides	123-133	myeloid differentiation primary response gene 88	Mus musculus	194-199	
16237045-4	2005	This defect in killing was due to diminished NADPH oxidase-mediated production of superoxide anion in response to bacteria by MyD88-/- phagocytes as a consequence of defective NADPH oxidase assembly.	Superoxides	82-98	myeloid differentiation primary response gene 88	Mus musculus	126-131	
19064699-0	2008	MyD88-dependent, superoxide-initiated inflammation is necessary for flow-mediated inward remodeling of conduit arteries.	Superoxides	17-27	myeloid differentiation primary response gene 88	Mus musculus	0-5