PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
30504838-8	2018	Neurons expressing either GluN2B subunits or chimeric GluN2A/GluN2B C-terminus subunits exhibited NMDA-induced superoxide production, whereas neurons expressing chimeric GluN2B/GluN2A C-terminus subunits did not.	Superoxides	111-121	glutamate receptor, ionotropic, NMDA2B (epsilon 2)	Mus musculus	61-67	
30504838-8	2018	Neurons expressing either GluN2B subunits or chimeric GluN2A/GluN2B C-terminus subunits exhibited NMDA-induced superoxide production, whereas neurons expressing chimeric GluN2B/GluN2A C-terminus subunits did not.	Superoxides	111-121	glutamate receptor, ionotropic, NMDA2B (epsilon 2)	Mus musculus	26-32	
30504838-8	2018	Neurons expressing either GluN2B subunits or chimeric GluN2A/GluN2B C-terminus subunits exhibited NMDA-induced superoxide production, whereas neurons expressing chimeric GluN2B/GluN2A C-terminus subunits did not.	Superoxides	111-121	glutamate receptor, ionotropic, NMDA2B (epsilon 2)	Mus musculus	61-67	
25158771-11	2014	CONCLUSIONS: Upregulation of NR2B phosphorylation at Y1472 after neonatal HI is involved in superoxide-mediated oxidative stress and contributes to brain injury.	Superoxides	92-102	glutamate receptor, ionotropic, NMDA2B (epsilon 2)	Mus musculus	29-33