PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
8523413-10	1995	In BCC, nicotine (1-10 mumol/l) evoked a release of NE and NPY and a transient rise of [Ca2+]i (determined with fura-2) during normoxia which were both dependent on the presence of extracellular calcium.	Nicotine	8-16	pro-neuropeptide Y	Cavia porcellus	59-62	
1378220-5	1992	Furthermore, the nicotine- but not capsaicin-evoked CGRP-LI release was inhibited by NPY.	Nicotine	17-25	pro-neuropeptide Y	Cavia porcellus	85-88	
1667328-3	1991	The nicotine-induced (100 mumol/l) overflow of noradrenaline and NPY was in a linear manner related (r = 0.79 and 0.90, respectively; p less than 0.05) to the extracellular calcium concentration (0-1.85 mmol/l), and it was prevented by calcium-free perfusion.	Nicotine	4-12	pro-neuropeptide Y	Cavia porcellus	65-68	
1667328-0	1991	Nicotine-induced release of noradrenaline and neuropeptide Y in guinea-pig heart: role of calcium channels and protein kinase C. The role of calcium, calcium influx through calcium channels, and activation of protein kinase C for the nicotine-induced release of noradrenaline and of the sympathetic co-transmitter neuropeptide Y (NPY) was investigated in the guinea-pig isolated perfused heart.	Nicotine	0-8	pro-neuropeptide Y	Cavia porcellus	46-60	
1667328-0	1991	Nicotine-induced release of noradrenaline and neuropeptide Y in guinea-pig heart: role of calcium channels and protein kinase C. The role of calcium, calcium influx through calcium channels, and activation of protein kinase C for the nicotine-induced release of noradrenaline and of the sympathetic co-transmitter neuropeptide Y (NPY) was investigated in the guinea-pig isolated perfused heart.	Nicotine	0-8	pro-neuropeptide Y	Cavia porcellus	314-328	
1667328-0	1991	Nicotine-induced release of noradrenaline and neuropeptide Y in guinea-pig heart: role of calcium channels and protein kinase C. The role of calcium, calcium influx through calcium channels, and activation of protein kinase C for the nicotine-induced release of noradrenaline and of the sympathetic co-transmitter neuropeptide Y (NPY) was investigated in the guinea-pig isolated perfused heart.	Nicotine	0-8	pro-neuropeptide Y	Cavia porcellus	330-333	
1667328-0	1991	Nicotine-induced release of noradrenaline and neuropeptide Y in guinea-pig heart: role of calcium channels and protein kinase C. The role of calcium, calcium influx through calcium channels, and activation of protein kinase C for the nicotine-induced release of noradrenaline and of the sympathetic co-transmitter neuropeptide Y (NPY) was investigated in the guinea-pig isolated perfused heart.	Nicotine	234-242	pro-neuropeptide Y	Cavia porcellus	46-60	
1667328-2	1991	In the presence of extracellular calcium (1.85 mmol/l) nicotine (1-100 mumol/l) evoked a concentration-dependent overflow of both transmitters with a molar ratio of approximately 1500 (noradrenaline):1 (NPY).	Nicotine	55-63	pro-neuropeptide Y	Cavia porcellus	203-206	
2784250-0	1989	Differential release of calcitonin gene-related peptide and neuropeptide Y from the isolated heart by capsaicin, ischaemia, nicotine, bradykinin and ouabain.	Nicotine	124-132	pro-neuropeptide Y	Cavia porcellus	60-74	
2784250-7	1989	Nicotine exposure induced release of CGRP- as well as NPY-LI in a concentration- and Ca2+-dependent manner.	Nicotine	0-8	pro-neuropeptide Y	Cavia porcellus	54-57	
2784250-12	1989	Activation of cardiac sensory nerves by capsaicin, nicotine, bradykinin and ouabain, as well as ischaemia, induced release of CGRP while nicotine also evoked NPY release.	Nicotine	137-145	pro-neuropeptide Y	Cavia porcellus	158-161