PMID-sentid Pub_year Sent_text comp_official_name comp_offsetprotein_name organism prot_offset 18157476-3 2007 Our previous experiments showed that with the single pulses evoking 80% of the maximal population spike (PS) amplitude, nicotine (10 mumol/L) induced LTP-like response in the hippocampal CA1 region. Nicotine 120-128 carbonic anhydrase 1 Homo sapiens 187-190 23785432-0 2013 Prenatal nicotine and maternal deprivation stress de-regulate the development of CA1, CA3, and dentate gyrus neurons in hippocampus of infant rats. Nicotine 9-17 carbonic anhydrase 1 Homo sapiens 81-84 22286796-5 2011 RESULTS: Our study shows that: a) kainic acid single administration increased the number of alpha-bungarotoxin insentive nicotinic receptors, b) nicotine was able to prevent such changes, c) repeated nicotine administration is capable to attenuate the damage of CA1 and CA3 areas of the hippocampus. Nicotine 145-153 carbonic anhydrase 1 Homo sapiens 262-265 18157476-4 2007 In the present study, the nicotinic acetylcholine receptor (nAChR) subtypes and relevant neurotransmitter releases involved in LTP-like response induced by nicotine were investigated by extracellularly recording the PS in the pyramidal cell layer in the hippocampal CA1 region in vitro. Nicotine 156-164 carbonic anhydrase 1 Homo sapiens 266-269 18157476-7 2007 The results suggest that noradrenaline release secondary to the activation of kappa-bungarotoxin-sensitive nAChRs participates in LTP-like response induced by nicotine in the hippocampal CA1 region. Nicotine 159-167 carbonic anhydrase 1 Homo sapiens 187-190 16627626-5 2006 The densities of copper and zinc distributions in a subfield of the hippocampus CA1 region are also reduced after nicotine treatment. Nicotine 114-122 carbonic anhydrase 1 Homo sapiens 80-83 17156388-0 2006 Chronic nicotine-induced switch in Src-family kinase signaling for long-term potentiation induction in hippocampal CA1 pyramidal cells. Nicotine 8-16 carbonic anhydrase 1 Homo sapiens 115-118 17156388-1 2006 Here, we show that chronic nicotine exposure induces changes in Src signaling for the modulation of N-methyl-D-aspartate receptor (NMDAR) function and LTP induction in CA1 pyramidal cells. Nicotine 27-35 carbonic anhydrase 1 Homo sapiens 168-171 16564510-0 2006 Nicotine reverses consolidated long-term potentiation in the hippocampal CA1 region. Nicotine 0-8 carbonic anhydrase 1 Homo sapiens 73-76 16564510-3 2006 The present study demonstrates that nicotine reverses stabilized LTP in the hippocampal CA1 region, providing the first evidence that consolidated LTP can be reversed. Nicotine 36-44 carbonic anhydrase 1 Homo sapiens 88-91 10773216-0 2000 Nicotine reverses GABAergic inhibition of long-term potentiation induction in the hippocampal CA1 region. Nicotine 0-8 carbonic anhydrase 1 Homo sapiens 94-97 15099681-9 2004 In a second series of studies, both acute and pre-exposure of cultures to (-)-nicotine (1-10 microM) significantly reduced NMDA toxicity in the CA1 region. Nicotine 74-86 carbonic anhydrase 1 Homo sapiens 144-147 15099681-10 2004 Co-administration of cultures to (-)-nicotine (1-10 microM) with 100 nM corticosterone prevented corticosterone"s exacerbation of subsequent CA1 insult. Nicotine 33-45 carbonic anhydrase 1 Homo sapiens 141-144 12649299-2 2003 We investigated the effect of nicotine and methyllycaconitine (MLA) on the toxicity of N-methyl-d-aspartate (NMDA) in the CA1 area of hippocampal slices. Nicotine 30-38 carbonic anhydrase 1 Homo sapiens 122-125 11388409-0 2001 Tetrodotoxin-sensitive enhancement of inhibition in CA1 pyramidal neurones by nicotine. Nicotine 78-86 carbonic anhydrase 1 Homo sapiens 52-55 11388409-2 2001 We report that nicotine, applied for 5-10 min at concentrations similar to those found during smoking (0.5-5 M), resulted in all CA1 pyramidal neurones in a marked, phasic and tonic increase in the frequency and amplitude of spontaneous inhibitory currents. Nicotine 15-23 carbonic anhydrase 1 Homo sapiens 129-132 31916029-4 2020 These processes are controlled by glutamatergic signaling of hippocampal pyramidal neurons within the CA1 region, suggesting actions of nicotine on glutamatergic transmission in this region if present prenatally. Nicotine 136-144 carbonic anhydrase 1 Homo sapiens 102-105