PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
27349231-6	2016	In turn, trichostatin A, an HDAC inhibitor, significantly reduced CCCTC-binding factor (CTCF) occupancy in the imprinting control region upstream of the H19 gene locus and subsequently downregulated the expression of H19.	trichostatin A	9-23	H19 imprinted maternally expressed transcript	Homo sapiens	153-156	
34726962-3	2022	The administration of the deacetylase inhibitors (DACi) panobinostat, trichostatin A and SAHA affected the cell viability of H295R monolayer and spheroids and induced the over-expression of H19 and autophagy transcripts.	trichostatin A	70-84	H19 imprinted maternally expressed transcript	Homo sapiens	190-193	
27349231-6	2016	In turn, trichostatin A, an HDAC inhibitor, significantly reduced CCCTC-binding factor (CTCF) occupancy in the imprinting control region upstream of the H19 gene locus and subsequently downregulated the expression of H19.	trichostatin A	9-23	H19 imprinted maternally expressed transcript	Homo sapiens	217-220	
25049758-8	2013	In the case of the imprinting genes H19 and NNAT, except PEG1, the SCNT blastocysts both with and without TSA treatment showed higher levels than those of the in vivo blastocysts.	trichostatin A	106-109	H19 imprinted maternally expressed transcript	Homo sapiens	36-39