PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26214583-4	2015	Interestingly, LPS increased the mRNA and protein levels of HDAC-4, -5, and -7, an effect that was abrogated by HDAC inhibitor trichostatin A (TSA) or TLR4-shRNA-lentivirus.	trichostatin A	127-141	histone deacetylase 4	Mus musculus	60-66	
31294671-4	2019	TSA is an inhibitor of class I and II histone deacetylases enzymes (HDAC), and for that, HDAC4 activity was determined.	trichostatin A	0-3	histone deacetylase 4	Mus musculus	89-94	
28234968-3	2017	We hypothesized that trichostatin A (TSA), a HDAC inhibitor, can reduce MV-augmented bleomycin-induced EMT by inhibiting the HDAC4 and Akt pathways.	trichostatin A	21-35	histone deacetylase 4	Mus musculus	125-130	
28234968-3	2017	We hypothesized that trichostatin A (TSA), a HDAC inhibitor, can reduce MV-augmented bleomycin-induced EMT by inhibiting the HDAC4 and Akt pathways.	trichostatin A	37-40	histone deacetylase 4	Mus musculus	125-130	
28234968-8	2017	CONCLUSIONS: Our data indicate that TSA treatment attenuates high-VT MV-augmented EMT after bleomycin-induced ALI, in part by inhibiting the HDAC4 and Akt pathways.	trichostatin A	36-39	histone deacetylase 4	Mus musculus	141-146	
26214583-4	2015	Interestingly, LPS increased the mRNA and protein levels of HDAC-4, -5, and -7, an effect that was abrogated by HDAC inhibitor trichostatin A (TSA) or TLR4-shRNA-lentivirus.	trichostatin A	143-146	histone deacetylase 4	Mus musculus	60-66	
25475100-6	2015	MG132, a potent proteasome pathway inhibitor, abrogated TSA-induced protective effects, which was associated with the accumulation of ubiquitinated HDAC4.	trichostatin A	56-59	histone deacetylase 4	Mus musculus	148-153	
25475100-8	2015	TSA treatment resulted in a decrease in HDAC4 in cardiomyocytes infected with adenoviral HDAC4, and HDAC4-induced injury was attenuated by TSA.	trichostatin A	0-3	histone deacetylase 4	Mus musculus	40-45	
25475100-8	2015	TSA treatment resulted in a decrease in HDAC4 in cardiomyocytes infected with adenoviral HDAC4, and HDAC4-induced injury was attenuated by TSA.	trichostatin A	0-3	histone deacetylase 4	Mus musculus	89-94	
25475100-8	2015	TSA treatment resulted in a decrease in HDAC4 in cardiomyocytes infected with adenoviral HDAC4, and HDAC4-induced injury was attenuated by TSA.	trichostatin A	0-3	histone deacetylase 4	Mus musculus	89-94	
21751234-3	2011	Using the well-established mouse ESC culture, we demonstrated that HDAC inhibitors, both trichostatin A (TSA,50  nmol/L) and sodium butyrate (NaB, 200  micromol/L) that causes the pronounced reduction of HDAC4 activity, decreased cell death and increased viability of ESCs in response to oxidant stress.	trichostatin A	89-103	histone deacetylase 4	Mus musculus	204-209	
21751234-3	2011	Using the well-established mouse ESC culture, we demonstrated that HDAC inhibitors, both trichostatin A (TSA,50  nmol/L) and sodium butyrate (NaB, 200  micromol/L) that causes the pronounced reduction of HDAC4 activity, decreased cell death and increased viability of ESCs in response to oxidant stress.	trichostatin A	105-108	histone deacetylase 4	Mus musculus	204-209