PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
12839289-9	2003	CONCLUSION: NOx concentrations increased throughout the neonatal HC+ sepsis period, reaching significant differences after 7-9 d. The highest ET-1 levels in neonatal HC+ sepsis emerged before the NOx peak, at 3-5 d, and later decreased.	nicotine 1-N-oxide	12-15	endothelin 1	Homo sapiens	142-146	
16170811-9	2005	The fact that NOx and ET-1 levels were higher in patients with decompensated cirrhosis (patients with ascites) than in those with compensated cirrhosis (patients without ascites), and the presence of a strong correlation between ET-1, NOx, and the degree of varices, supports the suggestion that there is a relationship between NOx, ET-1, and portal hypertension.	nicotine 1-N-oxide	14-17	endothelin 1	Homo sapiens	229-233	
16170811-9	2005	The fact that NOx and ET-1 levels were higher in patients with decompensated cirrhosis (patients with ascites) than in those with compensated cirrhosis (patients without ascites), and the presence of a strong correlation between ET-1, NOx, and the degree of varices, supports the suggestion that there is a relationship between NOx, ET-1, and portal hypertension.	nicotine 1-N-oxide	14-17	endothelin 1	Homo sapiens	229-233	
16170811-9	2005	The fact that NOx and ET-1 levels were higher in patients with decompensated cirrhosis (patients with ascites) than in those with compensated cirrhosis (patients without ascites), and the presence of a strong correlation between ET-1, NOx, and the degree of varices, supports the suggestion that there is a relationship between NOx, ET-1, and portal hypertension.	nicotine 1-N-oxide	235-238	endothelin 1	Homo sapiens	22-26	
16170811-9	2005	The fact that NOx and ET-1 levels were higher in patients with decompensated cirrhosis (patients with ascites) than in those with compensated cirrhosis (patients without ascites), and the presence of a strong correlation between ET-1, NOx, and the degree of varices, supports the suggestion that there is a relationship between NOx, ET-1, and portal hypertension.	nicotine 1-N-oxide	235-238	endothelin 1	Homo sapiens	22-26	
11393385-6	2001	While in control subjects significant correlations between urinary excretions of prostaglandin 6-keto F1alpha and thromboxane B2 (r = 0.69, p < 0.001) or NOx and ET-1 (r = 0.54, p < 0.001) were present, in patients only the relationship between urinary excretions of prostaglandin 6-keto F1alpha and thromboxane B2 (r = 0.53, p < 0.01) was retained.	nicotine 1-N-oxide	157-160	endothelin 1	Homo sapiens	165-169	
11508642-11	2001	There was a significant negative correlation between plasma NOx concentration and plasma ET-1 concentration.	nicotine 1-N-oxide	60-63	endothelin 1	Homo sapiens	89-93	
10551692-7	1999	RESULTS: Basal endothelin-1 levels were higher in CSX and MSX groups than in normal controls (8.19 +/- 0.46 and 6.97 +/- 0.88 vs. 3.67 +/- 0.99 pg/ml; p < 0.01), while basal NOx levels were significantly higher in MSX group than in CSX and normal controls (36.5 +/- 4.0 vs. 24.2 +/- 3.3 and 26.8 +/- 3.2 mol/liter, p < 0.05).	nicotine 1-N-oxide	177-180	endothelin 1	Homo sapiens	15-27	
11155153-2	2001	Increased levels of endothelin 1 (ET-1) and nitrites and nitrates (NOx, the end products of NO metabolism) have been reported in hepatocellular carcinoma (HCC), but the balance has not been studied.	nicotine 1-N-oxide	67-70	endothelin 1	Homo sapiens	20-32	
10983841-8	2000	There was a significant correlation between NOx (nitrite + nitrate) and ET-1 in sera from all 44 women (NP, NTP and PE groups) (p<0.001).	nicotine 1-N-oxide	44-47	endothelin 1	Homo sapiens	72-76