PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10203355-14	1999	It can be concluded that (1) iNOS can be induced without active NF-kappaB; (2) Dex, acetylsalicylic acid, and PDTC inhibit only p65; and (3) JAK2 is involved in iNOS induction, and the contribution of JAK2 to nitrite production is greater than that of NF-kappaB.	Aspirin	84-104	Janus kinase 2	Rattus norvegicus	141-145	
10203355-14	1999	It can be concluded that (1) iNOS can be induced without active NF-kappaB; (2) Dex, acetylsalicylic acid, and PDTC inhibit only p65; and (3) JAK2 is involved in iNOS induction, and the contribution of JAK2 to nitrite production is greater than that of NF-kappaB.	Aspirin	84-104	Janus kinase 2	Rattus norvegicus	201-205	
24836854-0	2014	Aspirin-triggered Lipoxin A4 attenuates mechanical allodynia in association with inhibiting spinal JAK2/STAT3 signaling in neuropathic pain in rats.	Aspirin	0-7	Janus kinase 2	Rattus norvegicus	99-103	
34307673-11	2021	Western blot analysis showed that compared with the MI/R group, aspirin pretreatment significantly increased the expression levels of p-STAT3 and p-JAK2 (p < 0.05).	Aspirin	64-71	Janus kinase 2	Rattus norvegicus	148-152	
34307673-12	2021	Conclusion: The mechanism of aspirin preconditioning to protect the heart from MI/R injury appears to be related to JAK2/STAT3 and related to the activation of the signaling pathway.	Aspirin	29-36	Janus kinase 2	Rattus norvegicus	116-120