PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
23766266-10	2013	CONCLUSIONS: Our results identify a novel proangiogenic role of LL-37, suggesting that the axis LL-37/COX-1/PGE2 followed by EP3 signaling is amenable to therapeutic intervention in pathological angiogenesis, for instance by aspirin.	Aspirin	225-232	prostaglandin E receptor 3	Homo sapiens	125-128	
23493387-0	2013	Prostaglandin receptor EP3 mediates growth inhibitory effect of aspirin through androgen receptor and contributes to castration resistance in prostate cancer cells.	Aspirin	64-71	prostaglandin E receptor 3	Homo sapiens	23-26	
23493387-3	2013	We also found that aspirin upregulated prostaglandin receptor subtype EP3 but not EP2 or EP4.	Aspirin	19-26	prostaglandin E receptor 3	Homo sapiens	70-73	
20587336-6	2010	Our findings provide further evidence that polymorphisms in PTGER3 might play a significant role in aspirin hypersensitivity among Korean asthmatics.	Aspirin	100-107	prostaglandin E receptor 3	Homo sapiens	60-66	
23493387-8	2013	Taken together, aspirin suppressed LNCaP cell proliferation via EP3 signaling activation; EP3 downregulation contributed to prostate carcinogenesis and to progression from androgen-dependent prostate cancer to castration-resistant prostate cancer by regulating AR expression.	Aspirin	16-23	prostaglandin E receptor 3	Homo sapiens	64-67