PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
10321245-4	1999	Chloroform, diethyl ether, halothane and isoflurane activated TREK-1, whereas only halothane and isoflurane activated TASK.	Chloroform	0-10	potassium two pore domain channel subfamily K member 2	Homo sapiens	62-68	
27397543-8	2016	Application of arachidonic acid (10 mumol/L), chloroform (1 mmol/L) or etomidate (10 mumol/L) substantially increased TREK-1 channel currents in CHO/hTREK-1 cells.	Chloroform	46-56	potassium two pore domain channel subfamily K member 2	Homo sapiens	118-124	
27397543-8	2016	Application of arachidonic acid (10 mumol/L), chloroform (1 mmol/L) or etomidate (10 mumol/L) substantially increased TREK-1 channel currents in CHO/hTREK-1 cells.	Chloroform	46-56	potassium two pore domain channel subfamily K member 2	Homo sapiens	149-156	
10880510-12	2000	As with TREK1, TREK2 is activated by the volatile general anesthetics chloroform, halothane, and isoflurane and by the neuroprotective agent riluzole.	Chloroform	70-80	potassium two pore domain channel subfamily K member 2	Homo sapiens	8-13	
32467161-3	2020	Here we show that inhaled anesthetics (chloroform and isoflurane) activate TREK-1 through disruption of phospholipase D2 (PLD2) localization to lipid rafts and subsequent production of signaling lipid phosphatidic acid (PA).	Chloroform	39-49	potassium two pore domain channel subfamily K member 2	Homo sapiens	75-81