PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
35170388-0	2022	Melatonin affects hypoxia-inducible factor 1alpha and ameliorates delayed brain injury following subarachnoid hemorrhage via H19/miR-675/HIF1A/TLR4.	Melatonin	0-9	H19 imprinted maternally expressed transcript	Homo sapiens	125-128	
27062045-0	2016	Long noncoding RNA H19 mediates melatonin inhibition of premature senescence of c-kit(+) cardiac progenitor cells by promoting miR-675.	Melatonin	32-41	H19 imprinted maternally expressed transcript	Homo sapiens	19-22	
27062045-8	2016	Interestingly, we found that long noncoding RNA H19 and its derived miR-675 were downregulated by H2 O2 in CPCs, but melatonin treatment could counter this alteration.	Melatonin	117-126	H19 imprinted maternally expressed transcript	Homo sapiens	48-51	
27062045-9	2016	Furthermore, knockdown of H19 or miR-675 blocked antisenescence actions of melatonin on H2 O2 -treated CPCs.	Melatonin	75-84	H19 imprinted maternally expressed transcript	Homo sapiens	26-29	
27062045-11	2016	In summary, melatonin antagonized premature senescence of CPCs via H19/miR-675/USP10 pathway, which provides new insights into pharmacological actions and potential applications of melatonin on the senescence of CPCs.	Melatonin	12-21	H19 imprinted maternally expressed transcript	Homo sapiens	67-70	
27062045-11	2016	In summary, melatonin antagonized premature senescence of CPCs via H19/miR-675/USP10 pathway, which provides new insights into pharmacological actions and potential applications of melatonin on the senescence of CPCs.	Melatonin	181-190	H19 imprinted maternally expressed transcript	Homo sapiens	67-70	
35170388-11	2022	Therefore, the treatment with MT could ameliorate post-SAH DBI.Running title: Melatonin ameliorates post-SAH DBI via H19/miR-675/HIF1A/TLR4 signaling pathways.	Melatonin	78-87	H19 imprinted maternally expressed transcript	Homo sapiens	117-120	
30240970-0	2018	Identifying Involvement of H19-miR-675-3p-IGF1R and H19-miR-200a-PDCD4 in Treating Pulmonary Hypertension with Melatonin.	Melatonin	111-120	H19 imprinted maternally expressed transcript	Homo sapiens	27-30	
30240970-0	2018	Identifying Involvement of H19-miR-675-3p-IGF1R and H19-miR-200a-PDCD4 in Treating Pulmonary Hypertension with Melatonin.	Melatonin	111-120	H19 imprinted maternally expressed transcript	Homo sapiens	52-55	
30240970-2	2018	The aim of this study was to characterize the therapeutic role of melatonin as well as the underlying molecular mechanism (its effects on the expression of H19 and its downstream signaling pathways) in the treatment of PAH.	Melatonin	66-75	H19 imprinted maternally expressed transcript	Homo sapiens	156-159	
30240970-9	2018	Finally, melatonin treatment inhibited cell proliferation; upregulated the expression of H19, miR-675-3p, and PDCD4; and downregulated the expression of miR-200a and IGF1R.	Melatonin	9-18	H19 imprinted maternally expressed transcript	Homo sapiens	89-92	
30240970-10	2018	This study demonstrated the role of H19-miR-675-3p-IGF1R- and H19-miR-200a-PDCD4-signaling pathways in the melatonin treatment of PAH.	Melatonin	107-116	H19 imprinted maternally expressed transcript	Homo sapiens	36-39	
30240970-10	2018	This study demonstrated the role of H19-miR-675-3p-IGF1R- and H19-miR-200a-PDCD4-signaling pathways in the melatonin treatment of PAH.	Melatonin	107-116	H19 imprinted maternally expressed transcript	Homo sapiens	62-65