PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
26497456-6	2016	These findings demonstrate that the decreased anabolism of glucose into UDPG and 1,3-beta-glucan triggered by a defect in the Rim15p-mediated upregulation of PGM2 and UGP1 redirects the glucose flux into glycolysis.	beta-1,3-glucan	81-96	protein kinase RIM15	Saccharomyces cerevisiae S288C	126-132