PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
34178389-4	2021	In settings of metabolic dysfunction, moieties such as excess sugars (glucose, fructose and sucrose) accumulate in the tissues and may form advanced glycation end products (AGEs), which are signaling ligands for the receptor for advanced glycation end products (RAGE).	Fructose	79-87	advanced glycosylation end-product specific receptor	Homo sapiens	216-260	
34178389-4	2021	In settings of metabolic dysfunction, moieties such as excess sugars (glucose, fructose and sucrose) accumulate in the tissues and may form advanced glycation end products (AGEs), which are signaling ligands for the receptor for advanced glycation end products (RAGE).	Fructose	79-87	advanced glycosylation end-product specific receptor	Homo sapiens	262-266	
31375034-12	2019	CONCLUSION: This study suggests that fructose may elicit endothelial cell damage partly via the activation of AGE-RAGE axis.	Fructose	37-45	advanced glycosylation end-product specific receptor	Homo sapiens	114-118	
30919933-10	2019	Instead, DC activation on acute exposure to fructose was via activation of receptor for advanced glycation end product (RAGE) in response to increased accumulation of advanced glycation end products (AGE).	Fructose	44-52	advanced glycosylation end-product specific receptor	Homo sapiens	75-118	
30919933-10	2019	Instead, DC activation on acute exposure to fructose was via activation of receptor for advanced glycation end product (RAGE) in response to increased accumulation of advanced glycation end products (AGE).	Fructose	44-52	advanced glycosylation end-product specific receptor	Homo sapiens	120-124