PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32765301-5	2020	High fat diet-fed C57BL/6J male mice and high-fat/high-glucose cultured Huh7 cells showed accumulation of both p62/SQSTM1 and LC3-II protein.	Glucose	55-62	sequestosome 1	Homo sapiens	111-114	
32765301-5	2020	High fat diet-fed C57BL/6J male mice and high-fat/high-glucose cultured Huh7 cells showed accumulation of both p62/SQSTM1 and LC3-II protein.	Glucose	55-62	sequestosome 1	Homo sapiens	115-121	
27345495-4	2016	Phosphorylation of p62/Sqstm1 at Ser349 directs glucose to the glucuronate pathway, and glutamine towards glutathione synthesis through activation of the transcription factor Nrf2.	Glucose	48-55	sequestosome 1	Homo sapiens	19-22	
27345495-4	2016	Phosphorylation of p62/Sqstm1 at Ser349 directs glucose to the glucuronate pathway, and glutamine towards glutathione synthesis through activation of the transcription factor Nrf2.	Glucose	48-55	sequestosome 1	Homo sapiens	23-29	
34657574-3	2021	In this study, it was found that SQSTM1/p62 (sequestosome 1), an autophagy receptor, was significantly downregulated in two human keratinocyte cells lines with short-term high-glucose treatment, as well as in the epidermis of diabetic patients and a db/db mouse model with long-term hyperglycemia.	Glucose	176-183	sequestosome 1	Homo sapiens	33-39	
34657574-3	2021	In this study, it was found that SQSTM1/p62 (sequestosome 1), an autophagy receptor, was significantly downregulated in two human keratinocyte cells lines with short-term high-glucose treatment, as well as in the epidermis of diabetic patients and a db/db mouse model with long-term hyperglycemia.	Glucose	176-183	sequestosome 1	Homo sapiens	40-43	
34657574-3	2021	In this study, it was found that SQSTM1/p62 (sequestosome 1), an autophagy receptor, was significantly downregulated in two human keratinocyte cells lines with short-term high-glucose treatment, as well as in the epidermis of diabetic patients and a db/db mouse model with long-term hyperglycemia.	Glucose	176-183	sequestosome 1	Homo sapiens	45-59	
28915571-8	2017	Indeed, p62 plasmid significantly reversed effects of HCD on the body mass index (BMI), levels of glucose, insulin and glycosylated hemoglobin (HbA1c).	Glucose	98-105	sequestosome 1	Homo sapiens	8-11	
26743088-0	2016	Regulation of glucose metabolism by p62/SQSTM1 through HIF1alpha.	Glucose	14-21	sequestosome 1	Homo sapiens	36-39	
26743088-0	2016	Regulation of glucose metabolism by p62/SQSTM1 through HIF1alpha.	Glucose	14-21	sequestosome 1	Homo sapiens	40-46	
26743088-8	2016	Functionally, HIF1alpha expression is required for p62-induced glucose uptake, lactate production and soft agar colony growth.	Glucose	63-70	sequestosome 1	Homo sapiens	51-54