PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32386481-6	2020	Furthermore, KLK10 suppression also afforded the suppressive effects on glycolysis in CRC cells as the evidences that targeting KLK10 restrained glucose uptake, lactate production and glycolysis-related glucose transporter 1 (Glut1) expression.	Glucose	145-152	kallikrein related peptidase 10	Homo sapiens	13-18	
32386481-6	2020	Furthermore, KLK10 suppression also afforded the suppressive effects on glycolysis in CRC cells as the evidences that targeting KLK10 restrained glucose uptake, lactate production and glycolysis-related glucose transporter 1 (Glut1) expression.	Glucose	145-152	kallikrein related peptidase 10	Homo sapiens	128-133	
26616394-4	2015	In vitro and in vivo assays showed that over-expressing KLK10 in PC3 could decelerate tumour proliferation, which was accompanied with an increase in apoptosis and suppression of glucose metabolism.	Glucose	179-186	kallikrein related peptidase 10	Homo sapiens	56-61	
26616394-8	2015	Thus, our results demonstrated that KLK10 may function as a tumour suppressor by repressing proliferation, enhancing apoptosis and decreasing glucose metabolism in PC3 cells.	Glucose	142-149	kallikrein related peptidase 10	Homo sapiens	36-41