PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
33307066-2	2021	Previously we reported that knockdown of the ubiquitously expressed ALAS1 gene in mice disrupts normal glucose metabolism, attenuates mitochondrial function and results in a prediabetic like phenotype when animals pass 20-weeks of age (Saitoh et al., 2018).	Glucose	103-110	aminolevulinic acid synthase 1	Mus musculus	68-73	
29364890-12	2018	This sequela is evidenced by a reduction of glucose uptake in C2C12 cells following the knockdown of ALAS1 or the inhibition of heme biosynthesis by succinylacetone.	Glucose	44-51	aminolevulinic acid synthase 1	Mus musculus	101-106	
22070747-3	2012	Acute porphyria attacks are usually treated by the administration of glucose; its effect is apparently related to its ability to inhibit ALAS1 by modulating insulin plasma levels.	Glucose	69-76	aminolevulinic acid synthase 1	Mus musculus	137-142