PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
31325222-7	2019	Participants with an impaired fasting glucose, high fasting glucose, and diabetes mellitus had higher mean levels of FVIII, FIX, and FXI than those with a normal glucose metabolism, with the highest differences in the levels of FVIII, FIX, and FXI between a high fasting glucose and a normal glucose metabolism.	Glucose	60-67	coagulation factor VIII	Homo sapiens	228-233	
32128578-0	2020	Factor VIII exhibits chaperone-dependent and glucose-regulated reversible amyloid formation in the endoplasmic reticulum.	Glucose	45-52	coagulation factor VIII	Homo sapiens	0-11	
32128578-6	2020	We show that FVIII forms amyloid-like fibrils within the ER lumen upon increased FVIII synthesis or inhibition of glucose metabolism.	Glucose	114-121	coagulation factor VIII	Homo sapiens	13-18	
32128578-7	2020	Significantly, FVIII amyloids can be dissolved upon restoration of glucose metabolism to produce functional secreted FVIII.	Glucose	67-74	coagulation factor VIII	Homo sapiens	15-20	
32128578-7	2020	Significantly, FVIII amyloids can be dissolved upon restoration of glucose metabolism to produce functional secreted FVIII.	Glucose	67-74	coagulation factor VIII	Homo sapiens	117-122	
31325222-5	2019	RESULTS: Per each mmol/L higher fasting glucose concentration we observed higher levels of fasting FVIII (5.33%, 95% CI: 4.00-6.65), FIX (6.19%, 95% CI: 5.15-7.23), and FXI (2.11%, 95% CI: 1.20-3.02).	Glucose	40-47	coagulation factor VIII	Homo sapiens	99-104	
31325222-7	2019	Participants with an impaired fasting glucose, high fasting glucose, and diabetes mellitus had higher mean levels of FVIII, FIX, and FXI than those with a normal glucose metabolism, with the highest differences in the levels of FVIII, FIX, and FXI between a high fasting glucose and a normal glucose metabolism.	Glucose	38-45	coagulation factor VIII	Homo sapiens	117-122	
31325222-7	2019	Participants with an impaired fasting glucose, high fasting glucose, and diabetes mellitus had higher mean levels of FVIII, FIX, and FXI than those with a normal glucose metabolism, with the highest differences in the levels of FVIII, FIX, and FXI between a high fasting glucose and a normal glucose metabolism.	Glucose	38-45	coagulation factor VIII	Homo sapiens	228-233	
31325222-7	2019	Participants with an impaired fasting glucose, high fasting glucose, and diabetes mellitus had higher mean levels of FVIII, FIX, and FXI than those with a normal glucose metabolism, with the highest differences in the levels of FVIII, FIX, and FXI between a high fasting glucose and a normal glucose metabolism.	Glucose	60-67	coagulation factor VIII	Homo sapiens	117-122	
31325222-7	2019	Participants with an impaired fasting glucose, high fasting glucose, and diabetes mellitus had higher mean levels of FVIII, FIX, and FXI than those with a normal glucose metabolism, with the highest differences in the levels of FVIII, FIX, and FXI between a high fasting glucose and a normal glucose metabolism.	Glucose	60-67	coagulation factor VIII	Homo sapiens	228-233	
31325222-7	2019	Participants with an impaired fasting glucose, high fasting glucose, and diabetes mellitus had higher mean levels of FVIII, FIX, and FXI than those with a normal glucose metabolism, with the highest differences in the levels of FVIII, FIX, and FXI between a high fasting glucose and a normal glucose metabolism.	Glucose	60-67	coagulation factor VIII	Homo sapiens	117-122	
31325222-7	2019	Participants with an impaired fasting glucose, high fasting glucose, and diabetes mellitus had higher mean levels of FVIII, FIX, and FXI than those with a normal glucose metabolism, with the highest differences in the levels of FVIII, FIX, and FXI between a high fasting glucose and a normal glucose metabolism.	Glucose	60-67	coagulation factor VIII	Homo sapiens	117-122	
31325222-7	2019	Participants with an impaired fasting glucose, high fasting glucose, and diabetes mellitus had higher mean levels of FVIII, FIX, and FXI than those with a normal glucose metabolism, with the highest differences in the levels of FVIII, FIX, and FXI between a high fasting glucose and a normal glucose metabolism.	Glucose	60-67	coagulation factor VIII	Homo sapiens	228-233	
31325222-7	2019	Participants with an impaired fasting glucose, high fasting glucose, and diabetes mellitus had higher mean levels of FVIII, FIX, and FXI than those with a normal glucose metabolism, with the highest differences in the levels of FVIII, FIX, and FXI between a high fasting glucose and a normal glucose metabolism.	Glucose	60-67	coagulation factor VIII	Homo sapiens	117-122	
31325222-7	2019	Participants with an impaired fasting glucose, high fasting glucose, and diabetes mellitus had higher mean levels of FVIII, FIX, and FXI than those with a normal glucose metabolism, with the highest differences in the levels of FVIII, FIX, and FXI between a high fasting glucose and a normal glucose metabolism.	Glucose	60-67	coagulation factor VIII	Homo sapiens	228-233	
31325222-9	2019	CONCLUSION: Concentrations of fasting glucose and HbA1c and postprandial glucose response were positively associated with FVIII, FIX, and FXI, and to some extent also with fibrinogen.	Glucose	38-45	coagulation factor VIII	Homo sapiens	122-127	
31325222-9	2019	CONCLUSION: Concentrations of fasting glucose and HbA1c and postprandial glucose response were positively associated with FVIII, FIX, and FXI, and to some extent also with fibrinogen.	Glucose	73-80	coagulation factor VIII	Homo sapiens	122-127	
25028444-7	2014	Adjusted for age, baseline stroke severity, and glucose, patients with FVIII+/vWF+ had increased odds of poor functional outcome (modified Rankin Scale>2; odds ratio, 2.87; 95% confidence interval, 1.16-7.06; P=0.021) than patients with FVIII-/vWF-.	Glucose	48-55	coagulation factor VIII	Homo sapiens	71-76	
27526421-10	2016	Independent predictors of FVIII >= 150% were: fibrinogen (p < 0.001), bilirubin (p = 0.002), hemoglobin (p = 0.016), glucose (p = 0.040), CRP (p = 0.023), total homocysteine (p = 0.032).	Glucose	123-130	coagulation factor VIII	Homo sapiens	26-31	
27526421-12	2016	CONCLUSIONS: The activity of FVIII in patients after VTE episode is influenced by age, concentration of fibrinogen, bilirubin, hemoglobin, glucose, CRP and homocysteine.	Glucose	139-146	coagulation factor VIII	Homo sapiens	29-34	
17504179-3	2007	The FVIII plasma activity is significantly associated with the carotid intima-media thickness and, strongly, with blood glucose and triglycerides levels.	Glucose	120-127	coagulation factor VIII	Homo sapiens	4-9	
8054845-5	1994	The bound FVIII was specifically dissociated from LCA-Sepharose by methyl-alpha-D-mannopyranoside, and to a lesser extent by other monosaccharides such as D-glucose, methyl-alpha-D-glucopyranoside, D-mannose, and D-galactose.	Glucose	155-164	coagulation factor VIII	Homo sapiens	10-15	
9525969-8	1998	In the presence of inhibitors of glucose trimming, the interactions of FVIII with CNX, and of FVIII and FV with CRT, were significantly reduced whereas the secretion of FVIII, and not FV, was inhibited.	Glucose	33-40	coagulation factor VIII	Homo sapiens	71-76	
9525969-8	1998	In the presence of inhibitors of glucose trimming, the interactions of FVIII with CNX, and of FVIII and FV with CRT, were significantly reduced whereas the secretion of FVIII, and not FV, was inhibited.	Glucose	33-40	coagulation factor VIII	Homo sapiens	94-99	
9525969-8	1998	In the presence of inhibitors of glucose trimming, the interactions of FVIII with CNX, and of FVIII and FV with CRT, were significantly reduced whereas the secretion of FVIII, and not FV, was inhibited.	Glucose	33-40	coagulation factor VIII	Homo sapiens	94-99