PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
32211094-0	2020	MiR-144 affects proliferation and apoptosis of high glucose-induced AC16 cardiomyocytes by regulating CTRP3/JNK signaling.	Glucose	52-59	microRNA 144	Homo sapiens	0-7	
32308422-13	2020	Functionally, FEZF1-AS1 silencing or miR-144 overexpression inhibited cell viability, the glucose and lactate productions and promoted cell apoptosis in Saos-2 and HOS cells.	Glucose	90-97	microRNA 144	Homo sapiens	37-44	
32308422-14	2020	Furthermore, miR-144 inhibitor mitigated the inhibitory effects on cell viability, the glucose and lactate productions and the promoted effect on cell apoptosis rate in Saos-2 and HOS cells induced by FEZF1-AS1 depletion.	Glucose	87-94	microRNA 144	Homo sapiens	13-20	
31292167-9	2019	In the present study, we focussed on a significantly down-regulated miRNA, miR-144-3p, and investigated its role in high glucose (HG) induced cell proliferation.	Glucose	121-128	microRNA 144	Homo sapiens	75-82	
31292167-15	2019	In conclusion, we demonstrated that miR-144-3p inhibited high glucose-induced cell proliferation through suppressing FGF16 and MAPK signaling pathway, suggesting a possible role of miR-144-FGF16 in the development of DR.	Glucose	62-69	microRNA 144	Homo sapiens	36-43	
31292167-15	2019	In conclusion, we demonstrated that miR-144-3p inhibited high glucose-induced cell proliferation through suppressing FGF16 and MAPK signaling pathway, suggesting a possible role of miR-144-FGF16 in the development of DR.	Glucose	62-69	microRNA 144	Homo sapiens	181-188	
27313692-6	2016	Glucose uptake rate and lactate production assays demonstrated that miR-144 expression is decreased and therefore enhances the aerobic metabolism in lung cancer cells.	Glucose	0-7	microRNA 144	Homo sapiens	68-75	
26175178-5	2016	The expression of platelet miR-144 and miR-223 significantly correlated with their plasma levels, P2Y12 and IRS-1 expression, blood glucose concentration, and platelet activation rate.	Glucose	132-139	microRNA 144	Homo sapiens	27-34	
26175178-6	2016	High glucose concentration significantly elevated P-selectin, miR-144 and P2Y12 expression and significantly reduced miR-223 and IRS-1 expression in UT-7 cells.	Glucose	5-12	microRNA 144	Homo sapiens	62-69	
26175178-7	2016	Overexpression of miR-223 and blocking of miR-144 expression significantly normalized the effects of high glucose concentration in UT-7 cells.	Glucose	106-113	microRNA 144	Homo sapiens	42-49	
27313692-7	2016	In addition, western blot analysis revealed that miR-144 performs this function by increasing the expression of glucose transporter 1 (GLUT1), leading to an increase in glucose uptake and lactate production.	Glucose	112-119	microRNA 144	Homo sapiens	49-56	
26662316-5	2016	As a result, cells overexpressing miR-144 exhibited a metabolic shift, including enhanced glucose uptake and lactate production.	Glucose	90-97	microRNA 144	Homo sapiens	34-41	
26662316-6	2016	The altered glucose metabolism induced by miR-144 also leads to a rapid growth of ovarian cancer cells.	Glucose	12-19	microRNA 144	Homo sapiens	42-49	
25237911-7	2014	Ectopic expression of miR-144/451a in melanoma cell lines resulted in markedly higher levels of mature miR-451a.1 than miR451a or miR-144; and significantly retarded cell migration and inhibited invasion in a glucose-sensitive manner.	Glucose	209-216	microRNA 144	Homo sapiens	22-29