PMID-sentid	Pub_year	Sent_text		comp_official_name	comp_offsetprotein_name	organism	prot_offset
24066113-4	2013	In the present study, we showed low glucose to induce a decrease of BCL2 and BCL-XL anti-apoptotic proteins expression, leading to an increase of free pro-apoptotic BAX.	Glucose	36-43	BCL2-like 1	Mus musculus	77-83	
22933114-0	2013	Bcl-2 and Bcl-xL suppress glucose signaling in pancreatic beta-cells.	Glucose	26-33	BCL2-like 1	Mus musculus	10-16	
22933114-2	2013	In this study, we demonstrate that chemical and genetic loss-of-function of antiapoptotic Bcl-2 and Bcl-x(L) significantly augments glucose-dependent metabolic and Ca(2+) signals in primary pancreatic beta-cells.	Glucose	132-139	BCL2-like 1	Mus musculus	100-105	
22933114-3	2013	Antagonism of Bcl-2/Bcl-x(L) by two distinct small-molecule compounds rapidly hyperpolarized beta-cell mitochondria, increased cytosolic Ca(2+), and stimulated insulin release via the ATP-dependent pathway in beta-cell under substimulatory glucose conditions.	Glucose	240-247	BCL2-like 1	Mus musculus	20-28	
22933114-6	2013	Inducible deletion of Bcl-x(L) in adult mouse beta-cells also increased glucose-stimulated NAD(P)H and Ca(2+) responses and resulted in an improvement of in vivo glucose tolerance in the conditional Bcl-x(L) knockout animals.	Glucose	72-79	BCL2-like 1	Mus musculus	22-30	
22933114-6	2013	Inducible deletion of Bcl-x(L) in adult mouse beta-cells also increased glucose-stimulated NAD(P)H and Ca(2+) responses and resulted in an improvement of in vivo glucose tolerance in the conditional Bcl-x(L) knockout animals.	Glucose	72-79	BCL2-like 1	Mus musculus	22-27	
22933114-6	2013	Inducible deletion of Bcl-x(L) in adult mouse beta-cells also increased glucose-stimulated NAD(P)H and Ca(2+) responses and resulted in an improvement of in vivo glucose tolerance in the conditional Bcl-x(L) knockout animals.	Glucose	162-169	BCL2-like 1	Mus musculus	22-30	
22933114-6	2013	Inducible deletion of Bcl-x(L) in adult mouse beta-cells also increased glucose-stimulated NAD(P)H and Ca(2+) responses and resulted in an improvement of in vivo glucose tolerance in the conditional Bcl-x(L) knockout animals.	Glucose	162-169	BCL2-like 1	Mus musculus	22-27	
12621302-0	2003	Bcl-XL maintains mitochondrial function in murine astrocytes deprived of glucose.	Glucose	73-80	BCL2-like 1	Mus musculus	0-6	
10662719-6	2000	However, Fd 2 Bcl-x(L) islets had impaired insulin secretory and intracellular free Ca(2+) ([Ca(2+)](i)) responses to glucose and KCl.	Glucose	118-125	BCL2-like 1	Mus musculus	14-22	
10662719-7	2000	Furthermore, insulin and [Ca(2+)](i) responses to pyruvate methyl ester (PME) were similarly reduced as glucose in Fd 2 Bcl-x(L) islets.	Glucose	104-111	BCL2-like 1	Mus musculus	120-128	
10662719-9	2000	Glucose-, PME-, and alpha-ketoisocaproate-induced hyperpolarization of mitochondrial membrane potential, NAD(P)H, and ATP production were also significantly reduced in Fd 2 Bcl-x(L) islets.	Glucose	0-7	BCL2-like 1	Mus musculus	173-181	
27070098-3	2016	We recently demonstrated that Bcl-2 and Bcl-xL, in addition to promoting survival, suppress beta-cell glucose metabolism and insulin secretion.	Glucose	102-109	BCL2-like 1	Mus musculus	40-46